Antidepressant Therapy

An, Crowson; Cm, Magro

doi:10.1001/archderm.1995.01690200063012

Cited by 63 publications

(2 citation statements)

References 24 publications

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“…Cutaneous lymphoid hyperplasia (CLH) is a reactive heterogeneous infiltration of benign T or B cell lymphocytes in response to a variety of stimulations, which simulate cutaneous malignant lymphoma clinically and histopathologically [1]. A variety of etiologies have been described, including tattoo dyes; vaccinations; metals used in piercings; infectious agents such as Borrelia burgdorferi, herpes simplex, herpes zoster, and molluscum contagiosum viruses; and drugs [2][3][4][5][6][7][8]. Because of epidemiological variations in infectious agents and cultural differences, the stimuli of CLH seem to be different in each country.…”

Section: Introductionmentioning

confidence: 99%

Clinical and Histopathological Characteristics of Cutaneous Lymphoid Hyperplasia: A Comparative Study According to Causative Factors

Choi

Lee

Lim

et al. 2020

JCM

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Cutaneous lymphoid hyperplasia (CLH) is a heterogeneous type of reactive lymphocytic infiltration resembling cutaneous lymphoma clinically and histopathologically. Few studies describe the relationship between the causative agents and histopathological and immunohistochemical characteristics of CLH. We investigated the clinical and histopathological characteristics of 50 patients with cutaneous CLH and analyzed them according to causative factors and predominant cell types (B or T cells). We retrospectively reviewed medical records to identify causative agents, and histopathological and immunohistochemical features. The majority of infiltrating lymphocytes were T cells (60%). T cell-dominant CLH showed papuloplaque lesions, whereas B cell-dominant CLH lesions were nodular. The infiltration pattern differed between T and B cells. In terms of prognosis, B-cell-predominant lesions tended to respond better to treatment than T-cell-predominant lesions. Hair dyes tended to be associated with multiple CLH lesions in older patients. CLH lesions associated with drugs were located on the head and neck. Insect bites were likely to cause a solitary papular lesion. Histopathologically, infiltration depth was located more superficially than other causes and featured intense eosinophilic infiltration. Thus, our study demonstrated that CLH presents different clinicopathological features according to causative agents and predominant lymphocytic types.

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Section: Introductionmentioning

confidence: 99%

Clinical and Histopathological Characteristics of Cutaneous Lymphoid Hyperplasia: A Comparative Study According to Causative Factors

Choi

Lee

Lim

et al. 2020

JCM

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“…Drug‐induced pseudolymphoma has been described with antiepileptic and antidepressant drugs, 25–28 methotrexate 29 and drugs with antihistamine properties 30,31 . The clinical presentation of drug‐induced pseudolymphoma may vary from a solitary asymptomatic erythematous and scaly nodule to an erythroderma resembling Sézary’s syndrome 25–31 .…”

Section: Discussionmentioning

confidence: 99%

Cutaneous pseudolymphoma, lymphomatoid contact dermatitis type, as an unusual cause of symmetrical upper eyelid nodules

Braun

French

Feldmann

et al. 2000

British Journal of Dermatology

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We describe a 74-year-old woman who rapidly developed bilateral voluminous nodules on the upper eyelids, following 4 months' treatment for chalazion. Histological and immunohistochemical findings were suggestive of cutaneous pseudolymphoma (CPL), and extensive screening for malignant lymphoma (ML) remained negative. Cutaneous pseudolymphomas are inflammatory diseases that can simulate ML either clinically, histopathologically, or both. They are a rare cause of nodules of the upper eyelids, usually characterized by a benign evolution, that can be secondary to ultraviolet sensitivity, adverse reactions to systemic medications and contact sensitization, among others. In our case, epicutaneous patch tests were strongly positive for a series of allergens contained in the ophthalmological preparations used; therefore, they were suggestive that the patient had CPL, lymphomatoid contact dermatitis type. In addition, the patient had been treated previously with several drugs, known to cause CPL and immune dysregulation. Complete regression of the lesions required treatment with systemic steroids and chlorambucil. No relapse occurred within a 5-year follow-up period.

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Drug-Induced Pseudolymphoma and Hypersensitivity Syndrome

Callot¹

1996

Arch Dermatol

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Antidepressant Therapy

Cited by 63 publications

References 24 publications

Clinical and Histopathological Characteristics of Cutaneous Lymphoid Hyperplasia: A Comparative Study According to Causative Factors

Clinical and Histopathological Characteristics of Cutaneous Lymphoid Hyperplasia: A Comparative Study According to Causative Factors

Cutaneous pseudolymphoma, lymphomatoid contact dermatitis type, as an unusual cause of symmetrical upper eyelid nodules

Drug-Induced Pseudolymphoma and Hypersensitivity Syndrome

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