“…Deshalb haben alle Bemiihungen, den Verlauf einer b6sartigen Erkrankung durch eine Ern/ihrungstherapie zu beeinflussen darauf abgezielt, entweder durch eine bestimmte Nfihrstoffzusammensetzung das Tumorwachsturn zu hemmen oder trotz einer m6glichen Steigerung des Tumorwachstums den Ernghrungszustand des Wirts zu verbessern [3,6]. Trotz der theoretischen M6glichkeit, das Carcinomwachstum durch Anti° metabolite oder den Entzug spezifischer Nfihrstoffe hemmen zu k6nnen, konnte eine direkte, ernfihrungsbedingte Manipulation der Tumorgr6Be in der klinischen Praxis nie nachgewiesen werden.…”
The importance of malnutrition as major cause of morbidity and mortality in cancer patients is well documented. It has been shown that nutritional therapy improves well-being and nutritional status of the patient, but the tumor can also be accelerated by hypercaloric nutrition. Our study does not confirm the induction of tumor growth. A high positive metabolic rate of the host lowers the tumor's growth rate and its tendency to spread. The retention of nitrogen in host increase with decreasing rate of energy and nutrition. Our results show that the tumor itself uses energy and nutrients from host cell catabolism and does not directly exploit exogenous nutrient substrates. It should not be forgotten that the animal model leads to specific changes affecting the energy and nutrient metabolism.
“…Deshalb haben alle Bemiihungen, den Verlauf einer b6sartigen Erkrankung durch eine Ern/ihrungstherapie zu beeinflussen darauf abgezielt, entweder durch eine bestimmte Nfihrstoffzusammensetzung das Tumorwachsturn zu hemmen oder trotz einer m6glichen Steigerung des Tumorwachstums den Ernghrungszustand des Wirts zu verbessern [3,6]. Trotz der theoretischen M6glichkeit, das Carcinomwachstum durch Anti° metabolite oder den Entzug spezifischer Nfihrstoffe hemmen zu k6nnen, konnte eine direkte, ernfihrungsbedingte Manipulation der Tumorgr6Be in der klinischen Praxis nie nachgewiesen werden.…”
The importance of malnutrition as major cause of morbidity and mortality in cancer patients is well documented. It has been shown that nutritional therapy improves well-being and nutritional status of the patient, but the tumor can also be accelerated by hypercaloric nutrition. Our study does not confirm the induction of tumor growth. A high positive metabolic rate of the host lowers the tumor's growth rate and its tendency to spread. The retention of nitrogen in host increase with decreasing rate of energy and nutrition. Our results show that the tumor itself uses energy and nutrients from host cell catabolism and does not directly exploit exogenous nutrient substrates. It should not be forgotten that the animal model leads to specific changes affecting the energy and nutrient metabolism.
“…Inhibition of carcinogenesis has an obvious bearing on cancer thresholds (9). Initial studies on inhibition date to the 1930's and represent some of the earliest experimental findings in carcinogenesis.…”
Section: Biologic Evidence For the Existence Of Thresholds Inmentioning
“…Remark on anticarcinoggenesis. Exposure of the host or target tissue t o a solitary carcinogen followed by one or more agents may result, not only in a n augmentation (" syncarcinogenesis ") but also in a n inhibition (" anticarcinogenesis "), of the carcinogenic response (for a review see Falk, 1971).…”
Section: Syncarcinogenesis (With a Remark On Anticarcinogenesis)mentioning
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