Aneurisma do Seio de Valsalva direito causando compressão coronariana extrínseca

Dias, Ricardo Ribeiro; Camurça, Flávio Duarte; Filho, Osanan Amorim Leite; Na, Stolf

doi:10.1590/s0066-782x2009000600017

Cited by 10 publications

(3 citation statements)

References 6 publications

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“…In previous studies, unruptured SVAs were generally asymptomatic and often presented as incidental findings during cardiac imaging, whereas those that ruptured into the cardiac chamber or pericardium induced acute heart failure or cardiac tamponade, respectively ( 9 ). Right SVAs protrude and rupture into the right ventricle, non-coronary SVAs tend to rupture into the right atrium, and left SVAs typically rupture into the pulmonary artery, left ventricle, or pericardium ( 10 ).…”

Section: Discussionmentioning

confidence: 99%

“…Blood stagnation in the SVA leads to embolization, which may migrate the coronary or peripheral arteries ( 11 ). Several reports have described that left or right SVAs cause compression of the LAD, while non-coronary SVAs cause compression of the RCA, which results in angina pectoris ( 9 , 12 , 13 ). In our patient, the dilated left SVA compressed the LM and proximal LCX during systole, inducing acute interruption of coronary blood flow, which was relieved during diastole.…”

Section: Discussionmentioning

confidence: 99%

“…The diagnosis and assessment of SVA relies on echocardiography, CCTA, and MRI ( 9 ). Three-dimensional images allow for a more accurate evaluation of aneurysms within the cardiac chambers and coronary arteries.…”

Section: Discussionmentioning

confidence: 99%

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Case report: Unruptured sinus of Valsalva aneurysms causing postural angina pectoris and false “pulmonary hypertension”

Zhou

Tao

2023

Front. Cardiovasc. Med.

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A 37-year-old woman presented with worsening intermittent chest pain and dyspnea in the previous year. Although the dyspnea was exertion-dependent, her chest pain was heavily dependent on her postural position, worsening in the supine position but alleviated by lying prone or by sitting up and leaning forward. She was cyanotic, and a diastolic murmur in the left third intercostal space was auscultated. An electrocardiogram recorded when she laid flat and had angina pectoris attacks showed ST-segment elevation in the aVR and depression in the II, III, aVF, and V3–V6 leads. However, when she sat up for a few minutes, her symptoms and ST-segment abnormalities disappeared. Echocardiography and cardiac computed tomography angiography revealed large unruptured aneurysms of the left and non-coronary sinuses, along with a dilated aortic root, severe aortic regurgitation, and right ventricular high pressure. Coronary angiography showed ∼90% pulsating stenosis of the left main coronary artery and ∼80% pulsating stenosis of the proximal left circumflex artery, presumably caused by pulsation of the dilated sinus of Valsalva aneurysm under blood pressure. Genetic testing revealed c.1781 C > G nonsense mutations in the FLNA gene. The patient underwent surgery, which confirmed dual unruptured left/non-coronary sinus of Valsalva aneurysms. Our case illustrates an unusual postural form of angina pectoris and false “pulmonary hypertension” caused by large dual unruptured left/non-coronary sinus of Valsalva aneurysms.

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Section: Discussionmentioning

confidence: 99%