Ambient carbon monoxide and the risk of cardiovascular disease emergency room visits: a time-series study in Lanzhou, China

You, Jinyuan; Liu, Yaocong; Dong, Jia-Yi; Wang, Jiancheng; Bao, Hairong

doi:10.1007/s10653-023-01653-1

Cited by 3 publications

(1 citation statement)

References 27 publications

Supporting

Mentioning

Contrasting

Order By: Relevance

“…Based on a study conducted in Lanzhou, China, the relative risks of daily ER visits were found to be 1.041 for total cardiovascular disease (CVD), 1.065 for ischemic heart disease (IHD), 1.083 for heart rhythm disturbances (HRD), 1.062 for heart failure (HF), and 1.057 for cerebrovascular diseases (CD) for every 1 mg/m3 increase in CO concentration [ 22 ]. Several investigations have examined the function of BNP and troponin in the diagnosis and prognosis of CO poisoning [ 17 , 21 , 23 ].…”

Section: Introductionmentioning

confidence: 99%

Cardiovascular biomarkers: exploring troponin and BNP applications in conditions related to carbon monoxide exposure

Taghdiri

2024

Egypt Heart J

View full text Add to dashboard Cite

Background The diagnosis and prognosis of cardiovascular disorders are greatly aided by cardiovascular biomarkers. The uses of troponin and B-type natriuretic peptide in situations involving carbon monoxide exposure are examined in this narrative review. These biomarkers are important because they help predict outcomes in cardiovascular disorders, track the effectiveness of therapy, and influence therapeutic choices. Main body Clinical practice makes considerable use of B-type natriuretic peptide (BNP), which has diuretic and vasodilatory effects, and troponin, a particular marker for myocardial injury. Carbon monoxide (CO) poisoning is a major worldwide health problem because CO, a “silent killer,” has significant clinical consequences. Higher risk of cardiac problems, poorer clinical outcomes, and greater severity of carbon monoxide poisoning are all linked to elevated troponin and B-type natriuretic peptide levels. BNP’s adaptability in diagnosing cardiac dysfunction and directing decisions for hyperbaric oxygen therapy is complemented by troponin’s specificity in identifying CO-induced myocardial damage. When combined, they improve the accuracy of carbon monoxide poisoning diagnoses and offer a thorough understanding of cardiac pathophysiology. Conclusions To sum up, this review emphasizes the importance of troponin and B-type natriuretic peptide (BNP) as cardiac indicators during carbon monoxide exposure. While BNP predicts long-term cardiac problems, troponin is better at short-term morbidity and death prediction. When highly sensitive troponin I (hsTnI) and B-type natriuretic peptide are combined, the diagnostic accuracy of carbon monoxide poisoning patients is improved. One of the difficulties is evaluating biomarker levels since carbon monoxide poisoning symptoms are not always clear-cut. Accurate diagnosis and treatment depend on the investigation of new biomarkers and the use of standardized diagnostic criteria. The results advance the use of cardiovascular biomarkers in the intricate field of carbon monoxide exposure.

show abstract

Section: Introductionmentioning

confidence: 99%

Cardiovascular biomarkers: exploring troponin and BNP applications in conditions related to carbon monoxide exposure

Taghdiri

2024

Egypt Heart J

View full text Add to dashboard Cite

show abstract

Influence of Air Pollution Exposures on Cardiometabolic Risk Factors: a Review

Guo,

Zeeshan,

Huang

et al. 2023

Curr Envir Health Rpt

View full text Add to dashboard Cite

Dietary Flavonoids: Mitigating Air Pollution’s Cardiovascular Risks

Rocha-Velasco,

Morales-Suárez-Varela,

Llopis-González

2024

Nutrients

View full text Add to dashboard Cite

Air pollution significantly impacts cardiovascular health, yet pollution reduction strategies in cardiovascular disease prevention remain limited. Dietary flavonoids show promise in protecting cardiovascular health, but their potential to mitigate air-pollution-induced risks is unexplored. This study investigates this research gap. Following PRISMA-ScR guidelines, literature from 2014–2024 was searched across MedLine/PubMed, ScienceDirect, and MDPI databases. Of 463 identified studies, 53 were eligible for analysis based on PICO criteria. Findings revealed significant impacts of air pollution on cardiovascular health, including increased disease risks and mortality. Flavonoid intake demonstrated protective effects against these risks. Flavonoid mechanisms include improved endothelial function, antioxidant and anti-inflammatory effects, blood pressure regulation, antiplatelet effects, cardioprotection, and enhanced lipid and glucose metabolism. Higher flavonoid intake was consistently associated with reduced cardiovascular risks. While reducing pollution remains crucial, promoting flavonoid-rich diets is a promising complementary strategy. Public health initiatives should raise awareness about these benefits. Further research on direct interactions between flavonoid intake and air pollution exposure is needed. Current evidence supports integrating dietary interventions into broader strategies to reduce air pollution’s cardiovascular impacts.

show abstract

Ambient carbon monoxide and the risk of cardiovascular disease emergency room visits: a time-series study in Lanzhou, China

Cited by 3 publications

References 27 publications

Cardiovascular biomarkers: exploring troponin and BNP applications in conditions related to carbon monoxide exposure

Cardiovascular biomarkers: exploring troponin and BNP applications in conditions related to carbon monoxide exposure

Influence of Air Pollution Exposures on Cardiometabolic Risk Factors: a Review

Dietary Flavonoids: Mitigating Air Pollution’s Cardiovascular Risks

Contact Info

Product

Resources

About