“…It is highly unlikely, however, for the factor in AD to be atrial natriuretic peptide, brain natriuretic peptide, or urodilatin because, in contrast to the findings in the present study, they increase sodium excretion principally by increasing glomerular filtration rate, and they are associated with a decrease in blood pressure 22 – 25 . Antidiuretic hormone, which can induce a natriuresis in rats, is not a likely factor in AD because low plasma levels, probably resulting from defective ADH secretion, are typical of AD 26 – 28 . It is also unlikely for the natriuretic effect of plasma to result from any of the medications taken by the patients, any associated illness, or differences in diet or sodium intake.…”