Air pollution and early deaths in the United States. Part I: Quantifying the impact of major sectors in 2005

Caiazzo, Fabio; Ashok, Akshay; Waitz, Ian A.; Yim, Steve Hung Lam; Barrett, Steven R. H.

doi:10.1016/j.atmosenv.2013.05.081

Cited by 354 publications

(233 citation statements)

References 16 publications

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“…5 Personal exposure to traffi c-related air pollution is a function of both ambient concentrations (a function of vehicle technology and other factors) and time activity patterns. [59][60][61][62][63] The IEA maintains a technical-economic database that includes detailed information on transport activity, vehicle activity, energy demand, and well-to-wheel greenhouse gas and pollutant emissions. 64 The IEA's Global Electric Vehicle Outlook report tracks sales of electric vehicles, and the International Council on Clean Transportation maintains a set of data tables, comparison charts, and a conversion tool for comparing passenger vehicle fuel standards.…”

Section: : Deployment Of Low-emission Vehicles and Access To Publimentioning

confidence: 99%

The Lancet Countdown: tracking progress on health and climate change

et al. 2017

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The Lancet Countdown: tracking progress on health and climate change is an international, multidisciplinary research collaboration between academic institutions and practitioners across the world. It follows on from the work of the 2015 Lancet Commission, which concluded that the response to climate change could be "the greatest global health opportunity of the 21st century". The Lancet Countdown aims to track the health impacts of climate hazards; health resilience and adaptation; health co-benefi ts of climate change mitigation; economics and fi nance; and political and broader engagement. These focus areas form the fi ve thematic working groups of the Lancet Countdown and represent diff erent aspects of the complex association between health and climate change. These thematic groups will provide indicators for a global overview of health and climate change; national case studies highlighting countries leading the way or going against the trend; and engagement with a range of stakeholders. The Lancet Countdown ultimately aims to report annually on a series of indicators across these fi ve working groups. This paper outlines the potential indicators and indicator domains to be tracked by the collaboration, with suggestions on the methodologies and datasets available to achieve this end. The proposed indicator domains require further refi nement, and mark the beginning of an ongoing consultation process-from November, 2016 to early 2017-to develop these domains, identify key areas not currently covered, and change indicators where necessary. This collaboration will actively seek to engage with existing monitoring processes, such as the UN Sustainable Development Goals and WHO's climate and health country profi les. The indicators will also evolve over time through ongoing collaboration with experts and a range of stakeholders, and be dependent on the emergence of new evidence and knowledge. During the course of its work, the Lancet Countdown will adopt a collaborative and iterative process, which aims to complement existing initiatives, welcome engagement with new partners, and be open to developing new research projects on health and climate change.

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Section: : Deployment Of Low-emission Vehicles and Access To Publimentioning

confidence: 99%

The Lancet Countdown: tracking progress on health and climate change

et al. 2017

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“…It comes at great monetary and human cost: in the 83 largest urban areas of the United States alone, the amount of wasted time and fuel caused by congestion has been placed at US$ 60 billion (1). At the same time, the World Health Organization has estimated that over one million deaths per year worldwide can be attributed to outdoor air pollution (2), which is to a large part caused by vehicular traffic (3). Further adverse effects include fatalities through road accidents and economic losses from missed business activities.…”

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confidence: 99%

Quantifying the benefits of vehicle pooling with shareability networks

Santi

Resta

Szell

et al. 2014

Proc. Natl. Acad. Sci. U.S.A.

655

493

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Taxi services are a vital part of urban transportation, and a considerable contributor to traffic congestion and air pollution causing substantial adverse effects on human health. Sharing taxi trips is a possible way of reducing the negative impact of taxi services on cities, but this comes at the expense of passenger discomfort quantifiable in terms of a longer travel time. Due to computational challenges, taxi sharing has traditionally been approached on small scales, such as within airport perimeters, or with dynamical ad hoc heuristics. However, a mathematical framework for the systematic understanding of the tradeoff between collective benefits of sharing and individual passenger discomfort is lacking. Here we introduce the notion of shareability network, which allows us to model the collective benefits of sharing as a function of passenger inconvenience, and to efficiently compute optimal sharing strategies on massive datasets. We apply this framework to a dataset of millions of taxi trips taken in New York City, showing that with increasing but still relatively low passenger discomfort, cumulative trip length can be cut by 40% or more. This benefit comes with reductions in service cost, emissions, and with split fares, hinting toward a wide passenger acceptance of such a shared service. Simulation of a realistic online system demonstrates the feasibility of a shareable taxi service in New York City. Shareability as a function of trip density saturates fast, suggesting effectiveness of the taxi sharing system also in cities with much sparser taxi fleets or when willingness to share is low.carpooling | human mobility | urban computing | maximum matching

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“…Current studies have indicated that inhalation of ambient particulate matter (PM) ranks as the ninth cause of overall disease (21). Well-established evidence that exposure to air pollution is linked with heart failure, myocardial infarction, lung dysfunction, lung cancer, and increased morbidity (5,6,14,16,20,22,24,31) has caused a worldwide need for policy change. Further research regarding air pollution exposure in susceptible populations, including those with obesity, existing heart disease, cystic fibrosis, and more sensitive states is needed, particularly concerning exposure during development.…”

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confidence: 99%

Early life exposure to air pollution induces adult cardiac dysfunction

Gorr

Velten²,

Nelin

et al. 2014

American Journal of Physiology-Heart and Circulatory Physiology

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Exposure to ambient air pollution contributes to the progression of cardiovascular disease, particularly in susceptible populations. The objective of the present study was to determine whether early life exposure to air pollution causes persistent cardiovascular consequences measured at adulthood. Pregnant FVB mice were exposed to filtered (FA) or concentrated ambient particulate matter (PM2.5) during gestation and nursing. Mice were exposed to PM2.5 at an average concentration of 51.69 μg/m3 from the Columbus, OH region for 6 h/day, 7 days/wk in utero until weaning at 3 wk of age. Birth weight was reduced in PM2.5 pups compared with FA (1.36 ± 0.12 g FA, n = 42 mice; 1.30 ± 0.15 g PM2.5, n = 67 P = 0.012). At adulthood, mice exposed to perinatal PM2.5 had reduced left ventricular fractional shortening compared with FA-exposed mice (43.6 ± 2.1% FA, 33.2 ± 1.6% PM2.5, P = 0.001) with greater left ventricular end systolic diameter. Pressure-volume loops showed reduced ejection fraction (79.1 ± 3.5% FA, 35.5 ± 9.5% PM2.5, P = 0.005), increased end-systolic volume (10.4 ± 2.5 μl FA, 39.5 ± 3.8 μl PM2.5, P = 0.001), and reduced dP/d t maximum (11,605 ± 200 μl/s FA, 9,569 ± 800 μl/s PM2.5, P = 0.05) and minimum (−9,203 ± 235 μl/s FA, −7,045 ± 189 μl/s PM2.5, P = 0.0005) in PM2.5-exposed mice. Isolated cardiomyocytes from the hearts of PM2.5-exposed mice had reduced peak shortening (%PS, 8.53 ± 2.82% FA, 6.82 ± 2.04% PM2.5, P = 0.003), slower calcium reuptake (τ, 0.22 ± 0.09 s FA, 0.26 ± 0.07 s PM2.5, P = 0.048), and reduced response to β-adrenergic stimulation compared with cardiomyocytes isolated from mice that were exposed to FA. Histological analyses revealed greater picro-sirius red-positive-stained areas in the PM2.5 vs. FA group, indicative of increased collagen deposition. We concluded that these data demonstrate the detrimental role of early life exposure to ambient particulate air pollution in programming of adult cardiovascular diseases and the potential for PM2.5 to induce persistent cardiac dysfunction at adulthood.

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Air pollution and early deaths in the United States. Part I: Quantifying the impact of major sectors in 2005

Cited by 354 publications

References 16 publications

The Lancet Countdown: tracking progress on health and climate change

The Lancet Countdown: tracking progress on health and climate change

Quantifying the benefits of vehicle pooling with shareability networks

Early life exposure to air pollution induces adult cardiac dysfunction

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