Adenomatoid odontogenic tumor (adenoameloblastoma)

Milobsky, L; Milobsky, Stanley A.; Miller, Gary M.

doi:10.1016/0030-4220(75)90379-5

Cited by 12 publications

(9 citation statements)

References 5 publications

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“…In whatever way the relationship arises, AOT may commence around the crown of a developing permanent tooth. At an earlier stage, it appears to be nothing more than an enlarged dental follicle radiographically [12,47,48,[54][55][56][57][58]. …”

Section: Extracoronal Variantmentioning

confidence: 99%

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Development and Growth of Adenomatoid Odontogenic Tumor Related to Formation and Eruption of Teeth

Ide

Mishima

Kikuchi

et al. 2011

Head and Neck Pathol

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The number of published cases of adenomatoid odontogenic tumor (AOT) has steadily increased, and about half were reported in Asian populations. Although the clinicopathologic profile of AOT has been extensively reported, factual knowledge of its pathogenesis is rudimentary at best, and there is controversy as to precisely which odontogenic epithelium it arises from. AOT is a successional tooth-associated lesion which develops during the mixed dentition. The permanent successor differs from its deciduous predecessor in that it has an eruptive pathway from the dental follicle to the gingiva, the gubernaculum dentis. With this background in mind, the present review focuses mainly on the early development of AOT. We successfully demonstrated a close spatial relationship between AOT and the gubernaculum dentis in a typical case. From further observations of the same AOT in which an enclosed permanent canine showed enamel hypoplasia, it is possible to pinpoint areas around the crown of a developing successional tooth in continuity with the gubernaculum dentis as starting points. In addition, we discuss the unifying histogenetic concept of three clinical variants, namely, pericoronal (follicular), extracoronal (extrafollicular), (see Article note) and peripheral.

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Section: Extracoronal Variantmentioning

confidence: 99%

“…[30] and [53] for specific details) [44,48,49,52,57]. Given time, a fully erupted tooth carries a residual AOT to the gingiva (peripheral variant) [44,47,53] or leaves it within the alveolar bone surface (extracoronal variant) [4,13,33,47,60,[71][72][73][74].…”

Section: Brief Summarymentioning

confidence: 99%

Development and Growth of Adenomatoid Odontogenic Tumor Related to Formation and Eruption of Teeth

Ide

Mishima

Kikuchi

et al. 2011

Head and Neck Pathol

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“…10 Radiographically, the lesion frequently looked like a dentigerous cyst or follicular cyst. [1][2][3][4][5][6][7][8][9][10][11][12][13][14][15][16][17][18][19][20] The radiolucency associated with the odontogenic adenomatoid tumour may extend more apically than the dentigerous cyst.…”

Section: 5mentioning

confidence: 99%

“…[1][2][3][4][5][6][7][8][9][10][11][12][13][14][15][16][17][18][19][20] Apart from a few exceptional cases 4,5 the tumour is associated with unerupted teeth. The unerupted teeth are usually canine or lateral incisors.…”

Section: It Was Stafne Who Identified the Adenomatoid Odontogenic Tummentioning

confidence: 99%

Adenomatoid odontogenic tumour (adenoameloblastoma). Case report and review of the literature

Dayı

Gürbüz

Bilge

et al. 1997

Australian Dental Journal

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“…Although there is no consensus for the treatment of AOT, the most common treatment option is enucleation and curettage. [1][2][3][4][5][6][7] We read the articles that Ide et al cited in their letter, and we have seen that in these documented orthodontic treatments for AOT, the authors preferred total excision of the tumor and preserved the teeth. [2][3][4][5][6][7][8][9][10][11][12] Ide et al also mentioned a report of subtotal excision (marsupialization) with eruption of the teeth.…”

mentioning

confidence: 99%