Acute hydrocephalus in a child with Mycoplasma Cerebellitis

Gayatri, NA; Tyagi, Atul; Mahadevan, Usha

doi:10.1016/j.braindev.2008.09.011

Cited by 10 publications

(8 citation statements)

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“…9 Acute cerebellitis is a rare complication of M pneumoniae infection, with only isolated case reports in the pediatric medical literature. 6,10 -16 Studies have shown that establishing a causal relationship between M pneumoniae infection and acute cerebellitis is often problematic because of difficulties in culturing the pathogen and the possibility of false-positive complement fixation titers (particularly for immunoglobulin M). In addition, coinfections have been reported, and the pathogen can persist in the throat for weeks.…”

Section: Discussionmentioning

confidence: 99%

Acute Cerebellitis in Children

et al. 2016

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Acute cerebellitis is a rare inflammatory condition. It may have a benign, self-limiting course or present as a fulminant disease resulting in severe cerebellar damage or even sudden death. We present the clinical, laboratory, and radiologic data in 9 children diagnosed with acute cerebellitis, who were identified by database search in our pediatric medical center from January 2000 to November 2014. The main presenting symptom was headache, and the main presenting sign was ataxia. Bilateral diffuse hemispheric involvement was the most common imaging finding at presentation. Mycoplasma pneumoniae was the most common infectious pathogen found. Treatment included steroids in all cases, antibiotics in 4, and intravenous immunoglobulins in 6. Six patients had a full recovery, and 3 had residual neurologic complications. Magnetic resonance imaging (MRI) is the modality of choice for diagnosis. The course of acute cerebellitis varies from a commonly benign and self-limiting disease to an occasionally fulminant disease, resulting in severe cerebellar damage or sudden death.

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Section: Discussionmentioning

confidence: 99%

Acute Cerebellitis in Children

et al. 2016

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“…CSF analysis is likely to reveal pleocytosis, and in rare cases, antibodies against an infectious agent have been demonstrated. 8 It must be mentioned that performing a spinal tap in the face of significant cerebellar edema can be life-threatening. Imaging can reveal abnormalities suggestive of edema in the cerebellum.…”

Section: Acute Cerebellitismentioning

confidence: 99%

Approach to Acute Ataxia in Childhood: Diagnosis and Evaluation

Sivaswamy¹

2014

Pediatr Ann

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Ataxia refers to motor incoordination that is usually most prominent during movement or when a child is attempting to maintain a sitting posture. The first part of the review focuses on the anatomic localization of ataxia -both within the nervous system and without -using a combination of historical features and physical findings. The remainder of the review discusses etiological considerations that vary depending on the age group under consideration. In infancy, certain specific diseases, such as opsoclonus myoclonus ataxia syndrome, must receive special mention because the underlying disease process may be amenable to surgical intervention. In the toddler-and school-age groups, certain conditions (such as stroke and acute cerebellitis) require immediate recognition and imaging, whereas others (such as post-infectious ataxia and concussion) require close follow-up. Finally, mention must be made of diseases outside of the central nervous system that can present with ataxia, such as Guillain-Barré syndrome.T he word ataxia is derived from the Greek word ataktos, which means "lack of order." Ataxia is characterized by disturbances in the voluntary coordination of posture and movement. In children, it is most prominent during walking (the sine qua non being a staggering gait with impaired tandem), but it can also be present during sitting or standing, or when the child is performing movements of the arms, legs, or eyes.This review focuses on the etiology and diagnostic considerations for acute ataxia, which for the purposes of this discussion refers to ataxia with a symptom evolution time of less than 72 hours.

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“…In another case, a 6-year-old girl was admitted to hospital with ataxia, and influenza A and influenza B were identifed in CSF with PCR [8]. However, there are some reports that present query AC cases with cerebellar signs, where viral RNA cannot be found in the cerebrospinal fluid [27][28][29]. For example, a case, reported by Ishikawa et al [9], presented with fever, headache, and truncal ataxia; and no viral nucleic fragment was detected in CSF.…”

Section: Discussionmentioning

confidence: 99%