Acute gastric dilatation

Byrne, John J.; Cahill, John

doi:10.1016/0002-9610(61)90229-x

Cited by 41 publications

(20 citation statements)

References 9 publications

Supporting

Mentioning

Contrasting

Order By: Relevance

“…In experimental animals, in order to produce ischemic necrosis, closure of the right and left gastric and gastroepiploic arteries together with at least 80% of the collaterals is required [2]. The important causes are postoperative complications [3, 4], anorexia nervosa and bulimia, psychogenic polyphagia, diabetes mellitus, trauma, electrolyte disturbances, gastric volvulus, and spinal conditions [1, 5–10]. In our case, the patient had binge eating 6 hours before development of symptoms.…”

Section: Discussionmentioning

confidence: 99%

Acute Dilatation, Ischemia, and Necrosis of Stomach without Perforation

Sahoo

Kumar

Jaiswal

et al. 2013

Case Reports in Surgery

View full text Add to dashboard Cite

Acute gastric dilatation can have multiple etiologies which may lead to ischemia of the stomach. Without proper timely diagnosis and treatment, potentially fatal events such as gastric perforation, haemorrhage, and other serious complications can occur. Here we present a 36-year-old man who came to the casualty with pain abdomen and distension for 2 days. Clinically, abdomen was asymmetrically distended more in the left hypochondrium and epigastrium region. Straight X-ray abdomen showed opacified left hypochondrium with nonspecific gaseous distension of bowel. Exploratory laparotomy revealed dilated stomach with patchy gangrene over lesser curvature and fundic area. About 4 litres of brownish fluid along with semisolid undigested food particles was sucked out (mainly undigested pieces of meat). Limited resection of gangrenous areas and primary repair were done along with feeding jejunostomy. Necrosis of the stomach was confirmed on histopathology. The patient recovered well and was discharged on the tenth postoperative day.

show abstract

Section: Discussionmentioning

confidence: 99%

Acute Dilatation, Ischemia, and Necrosis of Stomach without Perforation

Sahoo

Kumar

Jaiswal

et al. 2013

Case Reports in Surgery

View full text Add to dashboard Cite

show abstract

“…Although acute gastric dilatation most commonly develops in the postoperative period, it may also develop as a result of anorexia nervosa, bulimia, trauma, electrolyte disturbances and diabetes mellitus 1–4. The pathophysiology of acute gastric dilatation remains unclear but it has been suggested that reflex inhibition of gastric motor nerves with unopposed parasympathetic activity may lead to gastric dilatation, similar to that seen in colonic pseudo-obstruction.…”

Section: Discussionmentioning

confidence: 99%

“…The pathophysiology of acute gastric dilatation remains unclear but it has been suggested that reflex inhibition of gastric motor nerves with unopposed parasympathetic activity may lead to gastric dilatation, similar to that seen in colonic pseudo-obstruction. The majority of patients (90%) present with profuse vomiting5 followed by abdominal pain 4. Respiratory embarrassment due to splinting of the diaphragm and aspiration of gastric contents may occur and early placement of a nasogastric tube is essential in order to prevent these complications.…”

Section: Discussionmentioning

confidence: 99%

Gastric necrosis complicating acute gastric dilatation after Nissen fundoplication

2011

View full text Add to dashboard Cite

“…There have been many reports of acute gastric dilatation in people after anesthesia [ 1,3]. These investigators postulated that acute gastric dilatation occurred secondarily to paralytic ileus or gastric atony produced by the various anesthetic agents.…”

Section: Discussionmentioning

confidence: 99%

Acute Gastric Dilatation in Nonhuman Primates: Review and Case Studies

1982

View full text Add to dashboard Cite

Abstract. Acute gastric dilatation occurs sporadically in laboratory-housed nonhuman primates. Clinical histories often include chronic drug administration, food restriction, accidental overfeeding, and prior anesthesia. Monkeys may be found dead or may have clinical signs of colic, abdominal distention, and dyspnea. Death in untreated cases is due to impaired venous return and cardiopulmonary failure. Gastric distention with fermented gaseous ingesta and congestion of the abdominal viscera are the predominant lesions. The cause of acute gastric dilatation is unknown, but it probably is multifactorial. Two principal factors seem to be intragastric fermentation associated with Clostridium perfringens, and abnormal gastric function.Acute gastric dilatation occurs sporadically in laboratory-housed nonhuman primates and there is a high mortality rate in untreated cases. The cause of this condition has not been investigated in controlled laboratory studies, but the clinical syndrome and the lesions are well characterized [2,7,11,[13][14][15][16]. In this paper, reported cases of acute gastric dilatation in nonhuman primates are reviewed and compared to the condition in 2 1 monkeys necropsied at a university medical facility.Acute gastric dilatation was first reported in eight macaques in 1967 [2]. Subsequently, there have been cases in both Old and New World monkeys [4,7,9,11,[13][14][15][16]. The clinical syndrome and lesions of acute gastric dilatation are remarkably consistent in all reports, including ours (see Results). The pathogenesis of acute gastric dilatation has not been elucidated, but there seem to be several common predisposing factors: dietary manipulation, microbial fermentation of ingesta, and prior anesthesia. Intercurrent disease rarely is present, although there was one report of acute gastric dilatation in a pet Ateles geoffroyi that also had rickets [7].The quantity and composition of monkeys' diets are important in the pathogenesis of acute gastric dilatation. Many monkeys with a history of food restriction in conjunction with behavioral experiments develop the syndrome following subsequent ad libitum feeding or overfeeding 111, 14, 151. Changes in feeding practice over 126

show abstract

Acute gastric dilatation

Cited by 41 publications

References 9 publications

Acute Dilatation, Ischemia, and Necrosis of Stomach without Perforation

Acute Dilatation, Ischemia, and Necrosis of Stomach without Perforation

Gastric necrosis complicating acute gastric dilatation after Nissen fundoplication

Acute Gastric Dilatation in Nonhuman Primates: Review and Case Studies

Contact Info

Product

Resources

About