Acute ataxia of unknown origin in children

Klingman, Walter O.; Hodges, Richard G.

doi:10.1016/s0022-3476(44)80031-2

Cited by 26 publications

(7 citation statements)

References 9 publications

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“…The natural history of the disorder varies but it can evolve into a chronic disabling illness. An association is recognised between DES and malignancy, usually of neural crest origin [9,22,43]. Some theories of the pathogenesis of DES propose that it is always a para-neoplastic phenomenon similar to neurological syndromes seen in adults with an occult neoplasm [10,13].…”

Section: Introductionmentioning

confidence: 99%

Neurological sequelae of the dancing eye syndrome

1996

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Section: Introductionmentioning

confidence: 99%

Neurological sequelae of the dancing eye syndrome

1996

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“…Behavioural changes are frequently reported. Early case reports described periods of loss of consciousness as common but this has become less of a feature in recent years, perhaps reflecting a change in aetiology 4‐6,8,10,11 …”

Section: Discussionmentioning

confidence: 99%

Cerebellar Encephalitis Following Epstein‐barr Virus Infection

Pitts

Norris

1998

Int J Clinical Practice

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“…Batten's seminal work was followed in subsequent years by publication of many case series of children with acute cerebellar ataxia. [3][4][5][6][7][8] These early reports of acute cerebellar ataxia were noted to occur in association with a variety of illnesses such as scarlet fever, whooping cough, influenza, measles, polio, and varicella.…”

Section: Acute Cerebellar Ataxiamentioning

confidence: 99%

Acute Cerebellar Ataxia, Acute Cerebellitis, and Opsoclonus-Myoclonus Syndrome

Desai

Mitchell

2012

J Child Neurol

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Acute cerebellar ataxia and acute cerebellitis represent a process characterized by parainfectious, postinfectious, or postvaccination cerebellar inflammation. There is considerable overlap between these entities. The mildest cases of acute cerebellar ataxia represent a benign condition that is characterized by acute truncal and gait ataxia, variably with appendicular ataxia, nystagmus, dysarthria, and hypotonia. It occurs mostly in young children, presents abruptly, and recovers over weeks. Neuroimaging is normal. Severe cases of cerebellitis represent the other end of the spectrum, presenting with acute cerebellar signs often overshadowed by alteration of consciousness, focal neurological deficits, raised intracranial pressure, hydrocephalus, and even herniation. Neuroimaging is abnormal and the prognosis is less favorable than in acute cerebellar ataxia. Acute disseminated encephalomyelitis may be confused with acute cerebellitis when the clinical findings are predominantly cerebellar, but lesions on neuroimaging are usually widespread. Paraneoplastic opsoclonus-myoclonus syndrome is often initially misdiagnosed as acute cerebellar ataxia, but has very specific features, course, and etiopathogensis.

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Acute ataxia of unknown origin in children

Cited by 26 publications

References 9 publications

Neurological sequelae of the dancing eye syndrome

Neurological sequelae of the dancing eye syndrome

Cerebellar Encephalitis Following Epstein‐barr Virus Infection

Acute Cerebellar Ataxia, Acute Cerebellitis, and Opsoclonus-Myoclonus Syndrome

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