Acquired Ichthyosis Triggered by an Osseous Hemangiopericytoma: A Case Report and Review of the Literature

Patsatsi, Aikaterini; Kyriakou, Aikaterini; Karavasilis, Vasilios; Tsatsou, Fragkiski; Lazaridis, Georgios; Kalabalikis, Dimitrios; Sotiriadis, Dimitrios

doi:10.1159/000358294

Cited by 6 publications

(4 citation statements)

References 13 publications

Supporting

Mentioning

Contrasting

Order By: Relevance

“…Ichthyoses are classified into inherited and acquired forms. 1 AI is histopathologically similar to ichthyosis vulgaris with compact orthohyperkeratosis, including a reduced granular layer and diminished filaggrin expression. 1,2 In our case, the expression of filaggrin was markedly diminished, consistent with previous reports.…”

Section: Acquired Ichthyosis Disclosing Intravascular Large B-cell Lymphomamentioning

confidence: 96%

“…1 AI is histopathologically similar to ichthyosis vulgaris with compact orthohyperkeratosis, including a reduced granular layer and diminished filaggrin expression. 1,2 In our case, the expression of filaggrin was markedly diminished, consistent with previous reports. 2 AI is often associated with malignant and non-malignant diseases, particularly Hodgkin's lymphoma.…”

Section: Acquired Ichthyosis Disclosing Intravascular Large B-cell Lymphomamentioning

confidence: 96%

See 1 more Smart Citation

Acquired ichthyosis disclosing intravascular large B‐cell lymphoma

Hagiwara

Shinkuma

Yokoyama

et al. 2021

The Journal of Dermatology

View full text Add to dashboard Cite

LETTER TO THE EDITOR proliferate in systemic small blood vessels, resulting in microvascular occlusion in organs. 4 IVLBCL is characterized by the absence of lymph node enlargement and the presence of non-specific findings such as fever, anemia, and elevated serum LDH, which may delay the diagnosis. 4 Recently, the usefulness of random skin biopsy has been reported in the diagnosis of IVLBCL. 5 In our case, IVLBCL was incidentally identified by the skin biopsy of AI. IVLBCL

show abstract

Section: Acquired Ichthyosis Disclosing Intravascular Large B-cell Lymphomamentioning

confidence: 96%

Section: Acquired Ichthyosis Disclosing Intravascular Large B-cell Lymphomamentioning

confidence: 96%

Acquired ichthyosis disclosing intravascular large B‐cell lymphoma

Hagiwara

Shinkuma

Yokoyama

et al. 2021

The Journal of Dermatology

View full text Add to dashboard Cite

show abstract

“…In these cases, varying combinations of Curth's criteria for diagnosing paraneoplastic dermatoses may provide an objective framework for dermatologic assessment: (a) concurrent onset of cutaneous disease and internal malignancy, (b) appearance and resolution of skin disease and malignancy follow a parallel course, (c) the skin disease is not part of a genetic syndrome, (d) paraneoplastic process occurs frequently with specific malignancies, and (e) the cutaneous disease is uncommon in the general population. 23,24 Malignant tumours of various organ systems, multiple myeloma, myelodysplastic syndrome and tumour lysis syndrome have been reported with ichthyosiform lesions; however, no unifying pathomechanism has been determined 23,[25][26][27][28][29][30][31] (Table 1).…”

Section: Pa R a N Eopl Astic Disor De R Smentioning

confidence: 99%

Acquired ichthyosis, asteatotic dermatitis or xerosis? An update on pathoetiology and drug‐induced associations

Park

Saeidian

Youssefian

et al. 2022

Acad Dermatol Venereol

View full text Add to dashboard Cite

Acquired ichthyosis (AI) or conditions mimicking ichthyosis have been documented for well over a century. AI is limited to the skin, but can be the first presenting sign of an underlying disease or an adverse effect of select medications or environmental exposures. 1,2 Most heritable ichthyoses, with the exceptions of late-onset ichthyosis vulgaris (IV) and Refsum disease, typically manifest between the immediate postnatal period and 5 years of age. 2 AI, however, does not follow an obvious age distribution; rather, the appearance and disappearance of AI tend to coincide with onset and resolution of an inciting factor. Clinically, AI closely resembles IV with diffuse, symmetrically distributed, white-to-brown scales which can be confused with severe cases of xerosis or eczema (Figure 1a-c). 1 Histopathology of AI usually shows varying degrees of orthohyperkeratosis, a uniformly diminished or absent granular layer, and lack of dermal inflammation. 1,3 In patients with suspected AI, a detailed investigation into the clinical course typically does not yield personal or family history of ichthyosis, atopy, keratosis pilaris or other keratodermaassociated phenotypes seen with the heritable ichthyoses. 1,2 In this article, we provide an up-to-date overview of AI and the pathomechanisms suggested in the literature. While the associated clinical features in many cases of AI are unknown, detailed examples are provided for the medical conditions, medications and exposures with clinical importance. In

show abstract

“…1 Histologically, AI is similar to hereditary ichthyosis vulgaris since it usually presents with mild compact hyperorthokeratosis, reduced or absent granular layers and a reduced rete-papilla pattern. 3 AI has been linked to several diseases such as malignant, infectious, inflammatory, autoimmune, metabolic, nutritional and endocrine disorders, as well as drugs intake, including cimetidine, clofazimine, hydroxyurea, cholesterol-lowering agents, nicotinic acid, 3-hydroxy-3-methylglutaryl-coenzyme A reductase inhibitors, triparanol, allopurinol, butyrophenones. 1,3 We report the first case of AI during acitretin therapy.…”

mentioning

confidence: 99%