Accidental and Surgical Trauma and Wound Healing of the Eye

Domarus, D. von; Naumann, G. O. H.

doi:10.1007/978-1-4613-8525-7_5

Cited by 6 publications

(5 citation statements)

References 150 publications

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“…This indicates a firm adherence between the retina and the outer wall of the eye due to the tack implanta tion. The circumscript fibrovascular prolifer ation arising from the choroid has similarities to choroidal reactions following traumatic rup ture of the choroid [5]. This is not surprising as every tack implantation is necessarily accom panied by a small perforation of the choroid.…”

Section: Discussionmentioning

confidence: 91%

Histopathology Findings following Retinal Tack Implantation (With 1 color plate)

Daus

Völcker²,

Alexandridis³

et al. 1989

Ophthalmologica

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For the first time this report describes the histopathological findings 2 months after retinal-tack implantation in a human eye. A stainless-steel tack was implanted to seal a small retinal tear during vitrectomy because of severe proliferative diabetic retinopathy in a 38-year-old man. By gross examination the enucleated eye showed a total retinal detachment except at the site where the tack had been implanted. Histologically a fibrovascular tissue proliferation arising from the choroid had grown into the adjacent retina at the tack site. No inflammatory reaction, pigment epithelium proliferation or glial-cell proliferation attributable to the tack were observed.

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Section: Discussionmentioning

confidence: 91%

Histopathology Findings following Retinal Tack Implantation (With 1 color plate)

Daus

Völcker²,

Alexandridis³

et al. 1989

Ophthalmologica

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“…Some mice of both strains showed recurrent small corneal ulcerations, a well-established clinical finding in alkali-injured patients. 9 Rapid healing MRL mice showed a larger incidence of recurrence compared with B6 mice. The increased incidence of recurrent small corneal ulcerations in MRL mice may be associated with increased corneal epithelium at risk.…”

Section: Discussionmentioning

confidence: 96%

“…These processes lead to decreased corneal transparency. 9 Alkali-burned corneas seldom heal properly and show reduced corneal transparency. Prevention of scar tissue is an important clinical consideration after corneal injury.…”

mentioning

confidence: 99%

Accelerated Wound Healing of Alkali-Burned Corneas in MRL Mice Is Associated with a Reduced Inflammatory Signature

Ueno

Lyons

Burzenski

et al. 2005

Invest. Ophthalmol. Vis. Sci.

104

109

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Alkali-burned corneas heal faster and more completely in MRL mice than in B6 mice, by means of rapid reepithelialization, reduced inflammation, and reduced fibrosis. Reduced inflammation, including decreased neutrophil infiltrates and the lack of a robust proinflammatory gene expression signature correlates with the rapid healing. However, the rapid-healing phenotype is not intrinsic to MRL hematopoietic progenitor cells.

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“…Therefore, the damage to the pulp may be caused by physical stress directly added to the dentin and the pulpal tissue. Pulp injury may occur by the same mechanisms as traumatic injuries to soft tissues surrounded by bone, such as traumatic vitreum detachment (17) and traumatic brain hemorrhage (18). Cavitation and shear strain to the pulp must be taken into consideration (18).…”

Section: Discussionmentioning

confidence: 99%

Tissue reactions after experimental luxation injuries in immature rat teeth

Miyashin

Kato

Takagi

1991

Dental Traumatology

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Immature permanent teeth often develop complications after luxation injuries. The pathology and the degenerative processes of the complications, however, are not well understood. In this study, we histologically examined the long-term reactions of immature rat teeth after standardized luxation injuries delivered in our previously developed experimental model. The upper first molars of 3-week-old male Wistar rats weighing 50-60 g were pushed horizontally toward the palate to cause a constant amount of dislocation. The follow-up changes of the luxated molars were studied up to 56 days after the trauma. Soon after the trauma, the periodontium had irregular fibers, degenerated cells, hemorrhage and edema in various places. After 12 h, a cell free area appeared in the palatal periodontium. In the alveolar bone and on the surface of the root, resorption occurred one day and 5-7 days after injury respectively. Although the damage in the periodontium and alveolar bone was gradually repaired, the root resorption, especially in the palatal cervical region, continued throughout the experimental period. In the most cases, the dental pulp did not seem to be damaged immediately after the trauma. By the 3rd day after injury, however, displacement of odontoblasts into the dentinal tubules was observed in many teeth. Moreover, formation of irregular dentin was observed in some teeth after 7 days. These changes of the pulp tended to disappear in the later stages of the experiment.

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Accidental and Surgical Trauma and Wound Healing of the Eye

Cited by 6 publications

References 150 publications

Histopathology Findings following Retinal Tack Implantation (With 1 color plate)

Histopathology Findings following Retinal Tack Implantation (With 1 color plate)

Accelerated Wound Healing of Alkali-Burned Corneas in MRL Mice Is Associated with a Reduced Inflammatory Signature

Tissue reactions after experimental luxation injuries in immature rat teeth

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