A Twin‐Singleton Comparison of Problem Behaviour in 2‐3‐Year‐Olds

Oord, Edwin J. C. G. van den; Koot, Hans M.; Boomsma, Dorret I.; Verhulst, Frank C.; Orlebeke, J.F.

doi:10.1111/j.1469-7610.1995.tb01302.x

Cited by 110 publications

(23 citation statements)

References 16 publications

(5 reference statements)

Supporting

Mentioning

Contrasting

Unclassified

Order By: Relevance

“…Twins are more likely to be born premature or experience birth complications than singletons (Luke et al 1991; Spellacy et al 1990) and there is some evidence that premature birth and low birth weights are associated with precocious puberty (Ibanez et al 2000; Wehkalampi et al 2011). In general, however, studies have found that by early- to mid-childhood twins are generally representative of singletons on wide variety of physical and behavioral measures (Rutter and Redshaw 1991; Van den Oord et al 1995). Thus it is unclear to what extent twin status may affect puberty or hormone concentration.…”

Section: Discussionmentioning

confidence: 99%

Genetic and Environmental Contributions to Covariation Between DHEA and Testosterone in Adolescent Twins

et al. 2015

View full text Add to dashboard Cite

Although several studies have shown that pubertal tempo and timing are shaped by genetic and environmental factors, few studies consider to what extent endocrine triggers of puberty are shaped by genetic and environmental factors. Doing so moves the field from examining correlated developmentally-sensitive biomarkers toward understanding what drives those associations. Two puberty related hormones, dehydroepiandrosterone and testosterone, were assayed from salivary samples in 118 MZ (62 % female), 111 same sex DZ (46 % female) and 103 opposite-sex DZ twin pairs, aged 12–16 years (M = 13.1, SD = 1.3). Pubertal status was assessed with a composite of mother- and self-reports. We used biometric models to estimate the genetic and environmental influences on the variance and covariance in testosterone and DHEA, with and without controlling for their association with puberty, and to test for sex differences. In males, the variance in testosterone and pubertal status was due to shared and non-shared environmental factors; variation in DHEA was due to genetic and non-shared environmental factors. In females, variance in testosterone was due to genetic and non-shared environmental factors; genetic, shared, and non-shared environmental factors contributed equally to variation in DHEA. In males, the testosterone-DHEA covariance was primarily due to shared environmental factors that overlapped with puberty as well as shared and non-shared environmental covariation specific to testosterone and DHEA. In females, the testosterone-DHEA covariance was due to genetic factors overlapping with pubertal status, and shared and non-shared environmental covariation specific to testosterone and DHEA.

show abstract

Section: Discussionmentioning

confidence: 99%

Genetic and Environmental Contributions to Covariation Between DHEA and Testosterone in Adolescent Twins

et al. 2015

View full text Add to dashboard Cite

show abstract

“…1995). The socioeconomic status of the parents of the twins was somewhat higher than the level in the general Dutch population (Rietveld et al.…”

Section: Methodsmentioning

confidence: 99%

Assessment and Etiology of Attention Deficit Hyperactivity Disorder and Oppositional Defiant Disorder in Boys and Girls

et al. 2007

Self Cite

View full text Add to dashboard Cite

Attention deficit hyperactivity disorder (ADHD) and oppositional defiant disorder (ODD) are more common in boys than girls. In this paper, we investigated whether the prevalence differences are attributable to measurement bias. In addition, we examined sex differences in the genetic and environmental influences on variation in these behaviors. Teachers completed the Conners Teacher Rating Scale-Revised:Short version (CTRS-R:S) in a sample of 800 male and 851 female 7-year-old Dutch twins. No sex differences in the factor structure of the CTRS-R:S were found, implying the absence of measurement bias. The heritabilities for both ADHD and ODD were high and were the same in boys and girls. However, partly different genes are expressed in boys and girls.

show abstract

“…However, assortative mating for most personality traits has been found to be low in magnitude (Maes et al, 1998). Further, twins and singletons have generally been found to experience similar rates of psychiatric disorders and behavioral and emotional problems (Gjone & Novik, 1995; Moilanen et al, 1999; Simonoff et al, 1997; van den Oord et al, 1995). There are however, two ways in which twins differ from singletons: (i) lower birth weight and shorter gestational age (Plomin et al, 2001), and (ii) delayed language development (Rutter & Redshaw, 1991).…”

Section: Potential Moderators Of Genetic and Environmental Influences On Impulsivitymentioning

confidence: 99%

Genetic and environmental influences on impulsivity: A meta-analysis of twin, family and adoption studies

Bezdjian¹,

Baker

Tuvblad

2011

Clinical Psychology Review

133

114

View full text Add to dashboard Cite

A meta-analysis of twin, family and adoption studies was conducted to estimate the magnitude of genetic and environmental influences on impulsivity. The best fitting model for 41 key studies (58 independent samples from 14 month old infants to adults; N = 27,147) included equal proportions of variance due to genetic (0.50) and non-shared environmental (0.50) influences, with genetic effects being both additive (0.38) and non-additive (0.12). Shared environmental effects were unimportant in explaining individual differences in impulsivity. Age, sex, and study design (twin vs. adoption) were all significant moderators of the magnitude of genetic and environmental influences on impulsivity. The relative contribution of genetic effects (broad sense heritability) and unique environmental effects were also found to be important throughout development from childhood to adulthood. Total genetic effects were found to be important for all ages, but appeared to be strongest in children. Analyses also demonstrated that genetic effects appeared to be stronger in males than in females. Method of assessment (laboratory tasks vs. questionnaires), however, was not a significant moderator of the genetic and environmental influences on impulsivity. These results provide a structured synthesis of existing behavior genetic studies on impulsivity by providing a clearer understanding of the relative genetic and environmental contributions in impulsive traits through various stages of development.

show abstract

A Twin‐Singleton Comparison of Problem Behaviour in 2‐3‐Year‐Olds

Cited by 110 publications

References 16 publications

Genetic and Environmental Contributions to Covariation Between DHEA and Testosterone in Adolescent Twins

Genetic and Environmental Contributions to Covariation Between DHEA and Testosterone in Adolescent Twins

Assessment and Etiology of Attention Deficit Hyperactivity Disorder and Oppositional Defiant Disorder in Boys and Girls

Genetic and environmental influences on impulsivity: A meta-analysis of twin, family and adoption studies

Contact Info

Product

Resources

About