A new understanding of the effect of filler minerals on the precipitation of synthetic C–S–H

Abstract

The filler effect is the most important physical mechanism of mineral admixtures in the early hydration of cement whose chemical properties greatly affect the precipitation of C-S-H. In this study, calcite, strontianite, magnesite, dolomite, quartz, whewellite and whitlockite were selected as the fillers. The morphology and reaction kinetics of synthetic C-S-H precipitated on the surfaces of different fillers were studied via electron microscopy observations and electrical conductivity and ion concentration measurements. The precipitation rate of C-S-H has a positive correlation with the affinity of Ca 2+ for adsorption on the fillers, which can be explained by the nucleation barrier of C-S-H. Extremely ordered honeycomb-like morphology of the C-S-H is found on calcite and strontianite surfaces, while less regular leaf-like or honeycomb-like C-S-H are found on whewellite and whitlockite. The ordered C-S-H pattern is related to the lattice cleavage of the ionic compound filler.In the case of quartz, C-S-H prefers growth along the tangential direction, which is quite different from the normal-direction growth on ionic compounds. The in-plane growth of C-S-H on quartz is believed to be induced by a layer of loosely physically-adsorbed Ca 2+ .

Tailoring the interface assembly of mesoporous TiO2 on BTO film toward high-performance UV photodetectors

“…Moreover, it shows a typical H2 hysteresis loop at a relative pressure P/P 0 of 0.4-0.7, demonstrating that the mTO film has typical spherical pores with narrow pore windows. [35][36][37] Hence, compared with the open mesopores on the mTO surface, the mesopore size distribution with an average diameter of the film was only 5.5 nm as estimated by the Barrett-Joyner-Halenda (BJH) method (Fig. S1b, ESI †).…”

The role of RNA modification in hepatocellular carcinoma

“…YTHDF2 is negatively correlated with survival in HCC patients and YTHDF2 upregulates octamer-binding transcription factor 4 (OCT4) expression in an m6Adependent manner, promoting HCC progression (Zhang et al, 2020a). miRNA 145 (miR-145) regulates m6A expression levels by regulating the 3′-UTR of YTHDF2 mRNA in HCC cells to regulate m6A expression levels (Yang et al, 2017c).…”

YT521-B homology domain family proteins as N6-methyladenosine readers in tumors

“…Since the potential target genes regulated by YTHDF1 might be associated with amino acid degradation and lipid metabolism in the tumor cell cycle, abnormalities in these physiological functions could lead directly to the occurrence and progression of HCC. YTHDF2 also acts as an oncogene to promote the HCC lung metastasis and cancer stem cell (CSC) phenotype through increasing m6A methylation levels of OCT4 mRNA in the 5′untranslated region (UTR) and OCT4 protein expression (Zhang et al, 2020). In addition, YTHDF2 attaches to m6A sites to boost mRNA degradation.…”