“…Structural genes are carB and carRP (mutants showing an albino phenotype), and regulatory genes are crgA (mutant showing a carotene overproducing phenotype). Additionally, the white-collar family ( mcwc1a , mcwc1b , and mcwc1c: mutants showing altered phototropism, crgA -dependent regulation, and light-defective induction; respectively) were functionally studied, proposing a complex model to explain the regulation of the light responses in Mucorales [ 84 , 90 , 91 , 92 , 93 , 94 , 95 ]. Later, the RNAi machinery of M. lusitanicus was thoroughly studied, dissecting all the steps of the canonical pathway: the triggering and amplification (genes rdrp1 and rdrp2 , mutants showing RNAi-defective phenotypes) [ 64 , 69 ], the production of siRNAs (genes dicer1 and dicer2 , mutants showing RNAi-defective phenotypes) [ 65 , 89 ], and the targeting machinery (genes ago1 , ago2 , and ago3 , mutants showing RNAi-defective phenotypes)) [ 66 ].…”