2013
DOI: 10.7812/tpp/12-118
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A Clinician’s Guide to the Diagnosis and Management of Gallbladder Volvulus

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Cited by 31 publications
(76 citation statements)
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“…Such a clinical course should alert the clinician of the possibility of GBV. Leucocytosis and raised C reactive protein are the usual laboratory findings which suggest inflammatory response which lacks specificity 2. Liver function tests are usually normal as in our patient who had leucocytosis and had essentially normal liver function tests.…”
Section: Discussionmentioning
confidence: 54%
“…Such a clinical course should alert the clinician of the possibility of GBV. Leucocytosis and raised C reactive protein are the usual laboratory findings which suggest inflammatory response which lacks specificity 2. Liver function tests are usually normal as in our patient who had leucocytosis and had essentially normal liver function tests.…”
Section: Discussionmentioning
confidence: 54%
“…1,10 This case highlights a previously undescribed anatomical anomaly that may predispose to gallbladder torsion. 1,10 This case highlights a previously undescribed anatomical anomaly that may predispose to gallbladder torsion.…”
Section: Images For Surgeons E793mentioning
confidence: 87%
“…Review of the current literature shows that gallbladder volvulus commonly presents as an acute cholecystitis, although isolated cases of gallbladder torsion mimicking acute appendicitis have also been reported [6,8]. Since no single clinical, serologic, or radiographic finding is pathognomonic, this condition is often misdiagnosed as acute acalculous cholecystitis, making a correct preoperative diagnosis challenging [4,5,9]. According to a review by Reilly et al [4], a preoperative diagnosis of gallbladder torsion was made in 32 of 125 (26%) patients reported within the last 20 years and death as an outcome was reported in seven of the 113 patients presenting after 1991 representing a mortality rate of 6%.…”
Section: Discussionmentioning
confidence: 99%
“…In a review by Pottorf et al [5], absence of gallbladder fixation to the liver resulting in excessive mobilization ability of gallbladder, relaxation and atrophy of a previously normal mesentery in the elderly (also known as visceroptosis) causing mesenteric elongation and thinning, atherosclerosis of the cystic artery and a tortuous cystic duct, and congenital anomalies that predispose individuals to elongated mesenteries have been reported as possible explanations.…”
mentioning
confidence: 99%
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