“…The pathophysiology of CCS appears to be predominantly mechanical, due to an excessively long or mispositioned blind loop proximal to the anastomosis, which promotes food passage into this loop, increasing pressure, and causing dilatation of this segment, leading to the characteristic symptoms of the syndrome: fullness, pain, reflux, regurgitation, postprandial vomiting and, eventually, food intolerance and cachexia. [1][2][3][4][5][6][7][8] This condition, first referred to as "candy cane syndrome" in 2007, appears to be an underappreciated surgical complication, but it has been increasingly reported, likely due to the increase in gastric bypass surgery for obesity. [1,3,4,8] Some case reports and small series of CCS treatment by surgical resection of the dilated loop achieved good results, but also emphasised the technical complexity of revisional surgery due to adhesions, which limits the possibility of surgical treatment in fragile patients.…”