A Case of Folliculitis Decalvans Involving the Beard, Face and Nape

Karakuzu, Ali; Erdem, Teoman; Aktaş, Akın; Atasoy, Mustafa; Güleç, Ali

doi:10.1111/j.1346-8138.2001.tb00143.x

Cited by 24 publications

(20 citation statements)

References 6 publications

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“…The lesions generally tend to coalesce to form a single large patch in the vertex [3]. It can sometimes affect the beard, axillae, pubis, and inner thighs [44]. It usually lasts a long time and it is progressive, with multiple acute outbreaks, as reflected in our results.…”

Section: Discussionsupporting

confidence: 49%

Primary Scarring Alopecia: Clinical-Pathological Review of 72 Cases and Review of the Literature

Villablanca

Fischer

García-García

et al. 2017

Skin Appendage Disord

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Purpose of the Study: To analyze the epidemiologic, demographic, clinical, and histological characteristics of primary scarring alopecia (PSA) cases diagnosed over a 7-year period at the Department of Dermatology, Hospital Clinic, Barcelona, Spain. Procedures: Seventy-two patients diagnosed with PSA between 2006 and 2012 were included. Age, sex, ethnic group, clinical pattern, predominant histological infiltrate, final clinical diagnosis, time of onset, treatments used, and clinical evolution were evaluated and correlated. Results: The ethnic groups were distributed as follows: 93% European-Caucasian, 5% Mestizo-American, 1% oriental, and 1% Afro-American. Most cases were females (71%), and mean age was 51 ± 6 years. The follicular pattern was the most common, and the predominant inflammatory infiltrate was lymphocytic. Lichen planopilaris and frontal fibrosing alopecia were the main diagnoses. When correlating clinical aspects and histopathology, lymphocytic PSAs had a subacute onset and resulted in a nonchanging, more stable form, while neutrophilic PSAs had a more acute onset with an evolution of acute outbreaks. PSAs in a late stage with an absent/mild infiltrate had a subclinical onset and a slowly progressive or stable evolution. Conclusions: The PSAs are severe trichological conditions. Their high clinical and histopathological variability make them a diagnostic and therapeutic challenge. Message of the Paper: Knowing the clinical and histopathological aspects of PSAs should be of crucial importance to the dermatologist.

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Section: Discussionsupporting

confidence: 49%

Primary Scarring Alopecia: Clinical-Pathological Review of 72 Cases and Review of the Literature

Villablanca

Fischer

García-García

et al. 2017

Skin Appendage Disord

View full text Add to dashboard Cite

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“…The disease course is usually chronic and slowly progressive. 174,300 Intranasal carriage of S aureus, 7,290,304 and an elevated serum erythrocyte sedimentation rate 289,300 and antistaphyloccocal antibody titer 291,301 occasionally are noted.…”

Section: Folliculitis Decalvansmentioning

confidence: 97%

“…10 In rare cases, folliculitis decalvans can affect the beard, face, and nape of the neck. 300,303 Reports of extremity, axillary, and pubic involvement 291 may represent coincident or alternative diagnoses. The disease course is usually chronic and slowly progressive.…”

Section: Folliculitis Decalvansmentioning

confidence: 97%

“…7 With the introduction of rifampin and combined use of fusidic acid and zinc to the therapeutic arena, successful suppression and sustained disease-free remission, lasting from months to years after treatment withdrawal, are possible in some patients. [288][289][290][291]296,301,304,312,313 Bactericidal action with excellent intracellular penetration and pathogen eradication by rifampin and fusidic acid, 288,290,300,312,315 and immunomodulatory effects noted with all 3 agents 288-290,300,312 probably underlie this remarkable effect. Rifampin can also eliminate the carriage state of S aureus.…”

Section: Folliculitis Decalvansmentioning

confidence: 99%

“…174,199,[288][289][290][291][292] Host predisposition to infection due to systemic or local immune deficits and S aureus strainerelated properties have been cited as causal factors. [288][289][290]292 Several acquired and inherited immune disturbances have been associated with folliculitis decalvans 203,[292][293][294][295][296][297][298][299] ; however, the majority of affected patients have no demonstrable systemic 289,290,300,301 or lesional 289,290 immune abnormality. It may be that strain-specific virulence factors of S aureus, including superantigen production 288,290 and intracellular persistence after phagocytosis, 288,302 account for expression of disease.…”

Section: Folliculitis Decalvansmentioning

confidence: 99%

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