2.P.144 Lp(α) levels during acute infections

Lipoprotein (a) does not participate in the early acute phase response to training or extreme physical activity and is unlikely to enhance any associated immediate cardiovascular risk

“…8 Lp(a) has also been described as an acute phase protein, with a reported increase in response to a variety of acute stimuli including postoperative trauma, 9 myocardial infarction, and angina, [10][11][12] but not in rheumatoid arthritis 13 or during acute infection. 14 Min et al also proposed that any APR-Lp(a) response might be influenced by the apolipoprotein (a) (apo(a)) kringle 4 dependent phenotype present. 15 Thus, the involvement of Lp(a) in the APR is not clear cut, both in the extent and the latency of the response, notably after acute cardiac episodes, [10][11][12] and could also depend on the exact stimulus involved.…”