Pulmonary disease and the autonomic nervous system: a new pathophysiological mechanism for Lady Windermere syndrome

Bronchiectasis with Nocardia infection was designated as Lady Windermere-like syndrome: a retrospective study

“…All the patients included in the present study had a >3 months disease duration, and they showed repeated exacerbations of pulmonary symptoms prior to definitive therapy. Key imaging characteristics include exudation, consolidation, thin-walled cavitary lesions, opacities, centrilobular nodules, and bronchiectasis predominantly involving the right middle lobe and lingula [5] . There are, however, several notable differences between LWS and Nocardia infection.…”

“…Key imaging characteristics include exudation, consolidation, thin-walled cavitary lesions, opacities, centrilobular nodules, and bronchiectasis predominantly involving the right middle lobe and lingula. [ 5 ] There are, however, several notable differences between LWS and Nocardia infection. Although high-resolution computed tomography (HRCT) of chest revealed abdominal-type bronchiectasis (i.e., bronchiectasis in the right middle lobe and lingular segments) in both diseases, cylindrical bronchiectasis is dominant in the majority of patients with LWS, whereas the nocardiosis has roughly the same proportion of cystic and cylindrical bronchiectasis.…”