“…We noticed the occurrence of marked epidermal necrosis obscuring exogenous pigmentation, which is cited as the primary pathogenic mechanism. When pressed or crushed, diplopods tend to release chemical substances, such as quinones and hydrogen cyanide, that induce an erythemato-purpuric inflammatory process followed by prolonged residual hypo- and/or hyperpigmentation 1 . Hypopigmentation results from temporary intervention of the functional activity of the epidermo-melanic unit presenting increased melanin production at the epidermis basal layer, while confetti hyperpigmentation results from follicular melanin activity.…”