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The results indicate that (i) EtOH-induced activation of caspase-12 could be one of the underlying mechanisms of hepatocyte apoptosis; (ii) EtOH-induced cell apoptosis was alleviated via ResV (10 μM) by inhibiting ER stress and caspase-12 activation in a SIRT1-dependent manner; and (iii) SIRT1 activated indirectly by ResV (10 μM) attenuates EtOH-induced hepatocyte apoptosis partly through inhibiting PDE activity.

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Granzyme F induces a novel death pathway characterized by Bid-independent cytochrome c release without caspase activation

Shi

Wang

et al. 2009

Cell Death Differ

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Z Fan

FADD cleavage by NK cell granzyme M enhances its self-association to facilitate procaspase-8 recruitment for auto-processing leading to caspase cascade

The Resveratrol Attenuates Ethanol‐Induced Hepatocyte Apoptosis Via Inhibiting ER‐Related Caspase‐12 Activation and PDE Activity In Vitro

Granzyme F induces a novel death pathway characterized by Bid-independent cytochrome c release without caspase activation

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