The authors of this Communication wish to cite additional papers relevant to their work. Reports by Fornasiero et al. [23a] and Sun et al., [23c] both dealing with B,N-codoped TiO 2 , were inadvertently omitted from the references cited in this Communication. In those reports, nitrogen doping was realized not by thermal treatment route in the presence of an ammonia atmosphere, but by a wet-chemistry route, and no B À N bond was formed. The complete reference [23] is given below. [23] a)

Iodine-doped (I-doped) mesoporous titania with a bicrystalline (anatase and rutile) framework was synthesized by a two-step template hydrothermal synthesis route. I-doped titania with anatase structure was also synthesized without the use of a block copolymer as a template. The resultant titania samples were characterized by X-ray diffraction, Raman spectroscopy, Fourier transform infrared, nitrogen adsorption, transmission electron microscopy, X-ray photoelectron spectroscopy, and UV-visible absorption spectroscopy. Both I-doped titania samples, with and without template, show much better photocatalytic activity than commercial P25 titania in the photodegradation of methylene blue under the irradiation of visible light (>420 nm) and UV-visible light. Furthermore, I-doped mesoporous titania with a bicrystalline framework exhibits better activity than I-doped titania with anatase structure. The effect of rutile phase in titania on the adsorptive capacity of water and surface hydroxyl, and photocatalytic activity was investigated in detail. The excellent performance of I-doped mesoporous titania under both visible light and UV-visible light can be attributed to the combined effects of bicrystalline framework, high crystallinity, large surface area, mesoporous structure, and high visible light absorption induced by I-doping.

Highlights d Single-cell analyses reveal links between HSC heterogeneity and thrombocythemia d JAK2-mutant HSCs in ET show strong megakaryocyte (Mk) lineage priming d JAK2-mutant HSCs in ET are more sensitive to IFN signaling to Mk differentiation d JAK2-mutant HSCs in ET exhibit distinct signatures upon treatment