In this study, ZnO/Ni3ZnC0.7/C spheres were synthesized successfully via a simple method based on metal-organic frameworks (MOFs). The experimental results show that the reaction time has a great influence on the structure of the material. ZnO/Ni3ZnC0.7/C spheres with controlled solid and yolk-shell structures have been obtained by altering the reaction time. When applied as anode materials, both the solid and the yolk-shell ZnO/Ni3ZnC0.7/C composites present excellent electrochemical performance. In addition, it is worth mentioning that the yolk-shell structure composite's property is superior to that of the solid one's in terms of lithium storage. The stable reversible capacity of yolk-shell ZnO/Ni3ZnC0.7/C can be retained at 1002 mA h g(-1) at 500 mA g(-1) after completion of 750 cycles, and it also exhibits superior rate performance. In contrast, the solid ZnO/Ni3ZnC0.7/C under the same conditions of testing shows a reversible capacity of 824 mA h g(-1).
Spinal cord injury (SCI) interrupts long-projecting descending spinal neurons and disrupts the spinal central pattern generator (CPG) that controls locomotion. The intrinsic mechanisms underlying re-wiring of spinal neural circuits and recovery of locomotion after SCI are unclear. Zebrafish shows axonal regeneration and functional recovery after SCI making it a robust model to study mechanisms of regeneration. Here, we use a two-cut SCI model to investigate whether recovery of locomotion can occur independently of supraspinal connections. Using this injury model, we show that injury induces the localization of a specialized group of intraspinal serotonergic neurons (ISNs), with distinctive molecular and cellular properties, at the injury site. This subpopulation of ISNs have hyperactive terminal varicosities constantly releasing serotonin activating 5-HT1B receptors, resulting in axonal regrowth of spinal interneurons. Axon regrowth of excitatory interneurons is more pronounced compared to inhibitory interneurons. Knock-out of htr1b prevents axon regrowth of spinal excitatory interneurons, negatively affecting coordination of rostral-caudal body movements and restoration of locomotor function. On the other hand, treatment with 5-HT1B receptor agonizts promotes functional recovery following SCI. In summary, our data show an intraspinal mechanism where a subpopulation of ISNs stimulates axonal regrowth resulting in improved recovery of locomotor functions following SCI in zebrafish.
Flower-like NiO with a highly hierarchical porous structure was synthesized and used as a cathode material for Li-O batteries. The disordered porous structure and specific pathways for O and Li ions led to an outstanding cycling performance over 80 cycles at a high current density of 200 mA g.
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