No abstract

Budd-Chiari syndrome BCS caused by hepatic venous outflow obstruction due to occlusion of hepatic vein and/or the inferior vena cava IVC consists of a variety of clinical manifestations such as abdominal pain, hepatomegaly, ascite associated with acute or chronic liver dysfunction and portal hypertension. A large number of therapies have been introduced for BCS, both medical including diuretics, thrombolytics, and anticoagulants and surgical such as peritoneovenous shunt, direct correction of the venous obstruction, decompressive portosystemic venous shunt, and in end-stage liver disease, orthotopic liver transplantation. More recently percutaneous transluminal angioplasty PTA has been used to relieve the obstruction. PTA differs from surgery in that it can be used repeatedly because it is less invasive and safer. Possible complications of PTA include stent migration, occlusion due to thrombus formation or neointimal hyperplasia, pulmonary embolism, hemorrage and infection. We experienced one case with complication of stent migration into right ventricle after PTA for BCS.