Citizen scientists, along with satellite and ground-based sensors, have revealed a new arc boundary at subauroral latitudes.

There has been an exciting recent development in auroral research associated with the discovery of a new subauroral phenomenon called STEVE (Strong Thermal Emission Velocity Enhancement). Although STEVE has been documented by amateur night sky watchers for decades, it is as yet an unidentified upper atmosphere phenomenon. Observed first by amateur auroral photographers, STEVE appears as a narrow luminous structure across the night sky over thousands of kilometers in the east-west direction. In this paper, we present the first statistical analysis of the properties of 28 STEVE events identified using Time History of Events and Macroscale Interactions during Substorms (THEMIS) all-sky imager and the Redline Emission Geospace Observatory (REGO) database. We find that STEVE occurs about 1 hr after substorm onset at the end of a prolonged expansion phase. On average, the AL index magnitude is larger and the expansion phase has a longer duration for STEVE events compared to subauroral ion drifts or substorms. The average duration for STEVE is about 1 hr, and its latitudinal width is~20 km, which corresponds to~¼ of the width of narrow auroral structures like streamers. STEVE typically has an equatorward displacement from its initial location of about 50 km and a longitudinal extent of 2,145 km. Plain Language Summary Strong Thermal Emission Velocity Enhancement (STEVE) is anatmospheric phenomenon that manifests across the night sky as an extremely thin yet long ribbon of vibrant purple and white hues. Although STEVE has been well documented by amateur auroral photographers for several decades, the scientific community only recently stumbled upon this phenomenon. In this paper, we report on the first statistical analysis of STEVE's optical characteristics using ground-based all-sky imagery and examined satellite data to determine the geomagnetic conditions favorable for the formation of STEVE. Our results verify that STEVE is narrow in the north-south direction, but it extends over a wider east-west region. We have also determined that STEVE displaces southward over its lifetime in most observations. More interestingly, all 28 STEVE events identified in this study were observed at the end of a prolonged substorm expansion phase. More recently, Gallardo-Lacourt et al. (2018) analyzed data from the Polar-Orbiting Environmental Satellite (POES)-17 satellite for one STEVE event identified by Time History of Events and Macroscale Interactions GALLARDO-LACOURT ET AL. 9893

Little is currently known about the optical phenomenon known as Steve. The first scientific publication on the subject suggests that Steve is associated with an intense subauroral ion drift (SAID). However, additional inquiry is warranted as this suggested relationship as it is based on a single case study. Here we present eight occurrences of Steve with coincident or near‐coincident measurements from the European Space Agency's Swarm satellites and show that Steve is consistently associated with SAID. When satellite observations coincident with Steve are compared to that of typical SAID, we find the SAID associated with Steve to have above average peak ion velocities and electron temperatures, as well as extremely low plasma densities.

Embryogenesis in hydra includes a variable period of dormancy; and this period, as well as subsequent stages through hatching, takes place within a thick cuticle that hinders observation. Thus, although the early stages of development have been well-characterized qualitatively, the middle and later stages are only poorly understood. Here, we provide a detailed description of the stages of embryogenesis, including the time required to traverse each of the stages, and the changes that occur in the type and number of cells throughout the stages. The events of cleavage and gastrulation occur within the first 48 h. Cleavage is holoblastic and unipolar and leads to a single-layered coeloblastula. Gastrulation occurs by ingression and is followed by the deposition of the thick cuticle. Thereafter, during the variable period of dormancy ranging from 2-24 weeks, little occurs; the important events are the conversion of the outer layer into an ectoderm and the appearance of the interstitial cell lineage. During the last 2 days before hatching, the endoderm and gastric cavity form, while stem cells of the interstitial cell lineage proliferate and differentiate into neurons, nematocytes, and secretory cells. Finally, the cuticle cracks, and the hatchling enlarges and emerges from the cuticle as a functional animal. The formation of the gastric cavity and the hatching of the embryo are both explicable in terms of the osmotic behavior of the animal and the hydrostatic forces generated by this behavior. Characteristics of development that are common to hydra and triploblastic phyla are presented.

Toxoplasma gondii infection triggers host microtubule rearrangement and organelle recruitment around the parasite vacuole. Factors affecting initial stages of microtubule remodeling are unknown. To illuminate the mechanism, we tested the hypothesis that the parasite actively remodels host microtubules. Utilizing heat-killed parasites and time-lapse analysis, we determined microtubule rearrangement requires living parasites and is time dependent. We discovered a novel aster of microtubules (MTs) associates with the vacuole within 1 h of infection. This aster lacks the concentrated foci of gamma (γ)-tubulin normally associated with MT nucleation sites. Unexpectedly, vacuole enlargement does not correlate with an increase in MT staining around the vacuole. We conclude microtubule remodeling does not result from steric constraints. Using nocodazole washout studies, we demonstrate the vacuole nucleates host microtubule growth in-vivo via γ-tubulinassociated sites. Moreover, superinfected host cells display multiple γ-tubulin foci. Microtubule dynamics are critical for cell cycle control in uninfected cells. Using non-confluent monolayers, we show host cells commonly fail to finish cytokinesis resulting in larger, multinucleated cells. Our data suggest intimate interactions between T. gondii and host microtubules result in suppression of cell division and/or cause a mitotic defect, thus providing a larger space for parasite duplication.