No abstract

Background The effect of bracing over natural history of stable dysplastic hips is not well known. This multicenter randomized trial aimed at objectifying the effect of abduction treatment versus active surveillance in infants of 3 to 4 months of age. Methods Patients were randomized to either Pavlik harness or active surveillance group. Ultrasound was repeated at 6 and 12 weeks post randomization. the primary outcome was the degree of dysplasia using the Graf α-angle at 6 months of age. The measurement of the acetabular index (Ai) on plain pelvis X-rays was used to identify persistent dysplasia after 9 months and walking age (after 18 months). Findings The Pavlik harness group (n = 55) and active surveillance group (n = 49) were comparable for predictors of outcome. At 12 weeks follow-up the mean α-angle was 60.5° ± 3.8° in the Pavlik harness group and 60.0° ± 5.6° in the active surveillance group. (p = 0.30). Analysis of secondary outcomes (standard of care) showed no treatment differences for acetabular index at age 10 months (p = 0.82) and walking age (p = 0.35). Interpretation Pavlik harness treatment of stable but sonographic dysplastic hips has no effect on acetabular development. Eighty percent of the patients will have a normal development of the hip after twelve weeks. therefore, we recommend observation rather than treatment for stable dysplastic hips.

Therapeutic study, clinical case series: Level IV.

PurposeTo collect and describe data on the natural history of abnormal ultrasound (US) findings in hips of infants under six months of age to serve as a reference to the design of screening programmes and treatment algorithms in the care for children with hip dysplasia.MethodsA search in PubMed of the terms “DDH” and “ultrasound” was done to find hips with abnormal US findings that were not treated. In cases of multiple periods of follow-up, the classification of every period was evaluated separately (individual hip follow-up periods).ResultsData of 13 561 hips with 16 991 follow-up periods were collected and analyzed. Most quantifiable classifications and follow-up periods were according to Graf (14 876) and a minor number of the hips had follow-up periods with femoral head coverage (FHC) (2115). Normal development without treatment in the first six months was for Graf 2a between 89% and 98%, for Graf 2c between 80% and 100% and for clustered data Graf 2a to 2c between 80% and 97%. For Graf 3 hips more than 50% were reported to develop into normal hips without treatment. As for Graf 4 hips this percentage was reported below 50%. For children with an FHC less than 50%, normalization was reported between 78% and 100%.ConclusionThe natural history of developmental dysplasia of the hip (DDH) shows a benign course, especially in the well-centered hips. This outcome probably contributes to the fact that all studies on US screening of hips for detection of relevant DDH in order to improve outcomes of treatment are rated as substantially underpowered.

The purpose of this study is to correlate the measurement of anterior knee laxity using the Rolimeter with the functional outcome of anterior cruciate ligament (ACL) deficient knees. We tested 29 patients (12 males/17 females) with an average age of 33 years (range 19-47) that had been treated for ACL rupture, either by reconstruction or conservatively. The average follow-up at time of testing was 33 months (range 6-67).Functional outcome was graded by means of a questionnaire based on the IKDC score, the sports activity rating scale (SARS), the Cincinnati knee-rating system and the activities of daily living (ADL) scale, and by two functional knee tests (the one-leg hop test and the cross-over hop test). The anterior knee laxity was measured for both knees with the Rolimeter, and the side-to-side difference was calculated. We have found no correlation between the joint laxity and the functional outcome score.(P>0.05). When we compared both groups, we found a significant, though very low, correlation between the laxity and the functional questionnaire for the reconstructed group. (r=0.51, P=0.036). Therefore, the joint laxity measurement does not necessarily reflect the functional outcome of ACL-deficient knees. An explanation can be found in the importance of the proprioception and neuromuscular control in compensating the ACL-deficient knee.