No abstract

The objective was to conduct a systematic review on vagus indigestion in ruminants. This syndrome, caused by vagus nerve dysfunction and characterized by motility disorders of the pre-stomachs and abomasum, is categorized into four types, based on the location of the functional disorder: type I or failure in eructation, type II or failure in omasal transport, type III or failure in the pyloric flow and type IV or indigestion caused by advanced pregnancy. Due to its clinical and economic importance for cattle breeding, it is essential to approach this disease, aiming to expand knowledge and promote the correct diagnosis by veterinarians working in the field of internal medicine for ruminants.

This study aimed to carry out a retrospective study of vagal indigestion cases diagnosed in cattle admitted at the Garanhuns Bovine Clinic, Federal Rural University of Pernambuco campus. This syndrome, caused by dysfunctions of the vagus nerve and characterized by motility disorders of the pre-stomachs and abomasum, represented 5.5% (70/1279) of digestive cases diagnosed in a period of 10 years and had an unfavorable prognosis in 78.3% of cases. Type II vagal indigestion was the most prevalent, accounting for 40% of cases, followed by type I (24.3%) and types III and IV, which accounted for 18.6% and 10.0% of cases, respectively. Vagal indigestion in 67.1% (47/70) of cases occurred as a result of other illnesses, such as traumatic reticuloperitonitis (27.7%), lung diseases (12.8%), gastric impaction (10.6%), abomasal ulcer (10.6%), lymphosarcoma (6.4%), and liver abscesses (6.4%). Motor changes in the gastrointestinal tract, such as hypomotility, abdominal distension, and bloat, as well as their consequences, were the most frequent clinical signs. Laboratory, ultrasonographic, and anatomopathological alterations mainly originate from the primary illnesses present in each case. The approach of this disease is essential due to its clinical and economic importance for livestock to expand the knowledge of its etiopathogenesis, thus contributing to a more accurate diagnosis by veterinarians working in the field of internal medicine for cattle.

This paper describes the clinical, laboratory and anatomopathologic findings of sheep affected by abomasum compaction. Clinically, the presence of a firm structure located in the abomasal region stands out. The hematology and ruminal fluid analyze revealed neutrofilia with regenerative left shift, hyperfibrinogenimia, fauna and flora impairment and chloride concentration of (39,33 mEq/L) respectively. Macroscopically, a distended and compacted abomasum was observed with the presence of type IV ulcers and diffuse serofibrinous peritonitis. These results confirm the severity of the abomasum compaction in small ruminants. Therefore, guidelines related to animal feed management practices, especially in times of drought are necessary to reduce the occurency of this disease.