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Psychoemotional discomfort results in oxidative stress, as proven by many studies. One of the hypotheses states that this effect is realized through the sympathetic system. For example, acute and chronic emotional distress led to a decrease in the concentration of magnesium, an increase in malondialdehyde, and the depletion of the antioxidant potential in young people stressed by a military situation (Cernak et al., 2000). Increased concentrations of plasma myeloperoxidase, interleukin-6, and salivary cortisol in musicians on the following day of the premiere concert indicate that stress of public performance leads to oxidative stress (Pilger et al., 2014). Acute myocardial infarction in stressed rats (the stress was caused by daylight/darkness exposure, overcrowding, isolation, new hierarchy, tilting the cage, and restriction of water or food) was accompanied by hyperactivation of the sympathetic system, increased malondialdehyde and peroxynitrite, and decreased superoxide dismutase and glutathione (Mercanoglu et al., 2008). Whisker removal-induced

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