We analyzed the degradation features by measuring the capacitance–voltage characteristics after electrically aging blue thermally activated delayed fluorescence (TADF) organic light-emitting diodes (OLEDs). The measurement was investigated in terms of the hole transfer layer (HTL) and electron transfer layer (ETL) structures. For the HTL, three different materials—N,N′–bis(naphthalen–1–yl)–N,N′–bis(phenyl)–benzidine (NPB), 4,4′,4″-tris(carbazol–9–yl)triphenylamine (TCTA), and 1,3–bis(carbazol–9–yl)benzene (mCP)—were used at the HTL/emission layer (EML) interface; the TCTA/EML interface had the highest stability among the interfaces. For the ETL, bis [2–(diphenylphosphino)phenyl] ether oxide (DPEPO) without further dopants was used as an exciton blocking layer (ExBL) to effectively confine the excitons at the EML. However, DPEPO has low stability and carrier mobility. Therefore, 0, 10, and 40 nm-thick ExBL devices were investigated; it was found that the 0 nm-thick ExBL device was the most stable. However, the 10 nm-thick ExBL is essential to confine the excitons at the EML, which ensures a high EL performance.
Background and ObjectivesThis study was performed to determine the predictive factors for edge dissection ED and clinical significance of ED after coronary stenting. Materials and Methods The study group comprised 215 patients 243 lesions, mean age 59 years, 157 male in whom coronary stents were implanted between June, 1994 and June, 1998. By angiography, EDs were categorized into minor a very focal segment 5mm from the stent margin , major 5mm with prominent adventitial staining and 50 of lumen compromize , and acute closure. Results 1. ED occurred in 30 12.3 , minor 15, major 12 out of 243 lesions. Twelve of 30 EDs were located at the distal margin of the stent and occurred during high pressure. 2. Development of ED after stenting significantly correlated with severity of stenosis at the stent margin 30 , 19/30 vs. 33/213, p 0. 0001 , degree of angulation 45 , 16/30 vs. 48/213, p 0.0001 , and calcification in the lesion 2/30 vs. 4/213, p 0.02 . 3. There was no significant difference in clinical success rate between two groups 27/30 vs. 175/185, NS . 4. CRR in major and acute closure EDs n 12 were significantly higher in patients treated with repeated angioplasty than in patients treated with additional stents 5/6 vs. 1/8, p 0.02 . Conclusions EDs after coronary stenting are relatively common and lesion's characteristics such as severity of stenosis 30 at the stent margin, angulation 45 , and calcification of the lesion are predictive factors for EDs. EDs are not associated with early adverse clinical events. However, CRR was significantly higher in patients treated by repeated angioplasty in major and acute closure EDs.
Background An earlier index of reperfusion after thrombolytic therapy in patients with acute myocardial infarction is desirable to determine whether additional therapy is necessary to salvage the myocardium. Cardiac troponin-T has been developed as a new myocardial specific marker for myocardial injury and has been used for early assessment of reperfusion therapy. This study was performed to investigate the utility of cardiac troponin-T for assessment of reperfusion therapy using serial serum troponin-T and the rapid assay kit.Methods The study was comprised of 70 patients M F 64 6, mean age 56 2 year with acute myocardial infarction and reperfusion therapy was initiated within 6 hours after the onset of symtoms. Blood samples for CK and troponin-T were taken before thrombolysis and then 60, 90 minutes, 3, 6, 12, 24, 48, and 72 hours after thrombolysis. We compared successful reperfusion index of troponin-T
scite is a Brooklyn-based organization that helps researchers better discover and understand research articles through Smart Citations–citations that display the context of the citation and describe whether the article provides supporting or contrasting evidence. scite is used by students and researchers from around the world and is funded in part by the National Science Foundation and the National Institute on Drug Abuse of the National Institutes of Health.
hi@scite.ai
10624 S. Eastern Ave., Ste. A-614
Henderson, NV 89052, USA
Copyright © 2024 scite LLC. All rights reserved.
Made with 💙 for researchers
Part of the Research Solutions Family.