Although a state of mild hypotension may be beneficial to limit blood loss during spinal deformity corrective surgery, acute and/or prolonged hypotension may jeopardize spinal cord vascularity and should be avoided especially during surgical treatment of high-risk deformities such as kyphosis. Early warning by multimodality physiologic neuromonitoring appears to be a useful method to alert surgeons of the potentially devastating problem of hypotension-induced spinal cord dysfunction and allows immediate corrective actions.

The prevalence of unobtainable intraoperative SCM during PVCR was 17.9% (20 of 112). Postoperative transient paraplegia occurred exclusively in patients with no monitorable data as a result of angular kyphosis with acute progressive myelopathy. The rate of transient spinal cord deficits was significantly higher when there was no obtainable SCM (4 of 20 vs. 0 of 92 with SCM; p = .0008).

The prevalence of SCM changes during PVCR above the conus medullaris was 16.7%, mostly during osteotomy and rod/screw compression. Data returned with prompt intervention and all had intact lower extremity motor function postoperatively. These SCM "saves" strongly emphasize the importance of multimodality neurophysiologic monitoring during high-risk cases, minimizing postoperative complications.