This article argues that Tahmima Anam’s novel The Good Muslim highlights the troubled relationship between empathy and silence in order to demonstrate the ethical dangers arising from the failure to accept “partial empathy”, wherein understanding and sharing coexists with incomprehension and disjunction. In doing so, the novel critiques both extremes of the theoretical debate about empathy: the dismissal of empathy, drawing on the Levinasian–Derridean discourse of alterity, for ineluctably subsuming the other’s otherness, and the celebration of empathy as a conciliatory bridge across polarizing differences. Moreover, Anam underlines the ambivalences of silence — which can embody both powerlessness and agency, and which can both participate in and withhold expression — to complicate the current emphasis on silencing in critical theory with concerns about the violation of others’ silences. Examining the silences of trauma and rape in the aftermath of the 1971 Bangladesh War, the novel shows how false empathy silences the other by deeming their experience as already understood while intrusive empathic gestures jeopardize the other’s chosen silences. Further, Maya and Sohail’s negotiation of their irreconcilable differences over religion illustrates how their inability to accept gaps in empathy prompts them to adopt conflict-eschewing silences that lead to the complete breakdown of empathy.
Background: To determine if mitotic activity played a role in classifying breast cancer in terms of its biological behaviour. We investigated the prospect of identifying a more meaningful cell proliferation marker for categorising treatment-naive breast cancer.
Methods: The 150 cases diagnosed as invasive breast carcinoma in the histopathology section were systematically studied for the clinical, gross, and microscopic features.
Results: the 50% patients were grade 2 (75), 41% were grade 3 (71) and 9% (14) were grade 1 in present study. The distribution of intrinsic subtypes was luminal A 25% (38), luminal B 59% (88), HER2 enriched 10% (15), basal 6% (9). Out of 150 cases, 29% (43) cases were T1, T2 were 65% (97), T3 were 2% (4), T4 were 4% (6). Mean Ki 67 was 15.6±8.8 in grade 1, 23.3±15.4 and 38.2± in grade 3. There was significant difference between I and III, and II and III (p<0.05). Mean mitotic count in grade 1 was 5.4±2.7, in grade 2 it was 9.7±13.5, in grade 3 it was 16.1±6.9. There was significant difference between grade 1 and 2, grade 2 and 3, grade 1 and 3 (p<0.05). There was significant difference between T stages (p<0.05).
Conclusions: Ki 67 showed a more significant statistical correlation with prognostic factors as compared to mitotic count; we feel Ki 67 is more superior to mitotic count as a prognostic factor.
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