Background
There is an opinion that Helicobacter Pylori may contribute to the progression of gastropathy. The presence of Helicobacter Pylori infection can accelerate the damage of gastric and duodenal mucosa. Gastropathy is mucosal disturbances that usually accompany with gastritis gastric ulcer, duodenal ulcer and gastric cancer. Endoscopically, the mucosal appearances consisted of mosaic pattern, adema, hyperemia, superficial ulceration and red spot.
Objective
To assess the influence of Helicobacter Pylori on gastropathy.
Methods
The study enrolled a total 50 patients with gastroscopic lesions (gastritis, gastric ulcer, duodenal ulcer, gastric cancer) and positive CLO test, to undergo evaluation on gastropathic appearences.
Result
Reported in the third western pacific Helicobacter congress June 22‐24 Bali Indonesia
Background
Helicobacter pylori (HP) can colonise beneath layer of mucous on the surface of gastric epithelial cells caused chronic infection more than 95% at antrum (chronic antral inflamation) besides corpus with spreading inequitably. This inflamation can cause gastric lession as a Gastritis, Gastric Ulcer (GU), Cancer of Duodenal Ulcer (DU). Absence of chronic antral inflamation (increased number of mononuclear cells in lamina propia) is a highly specific methode to exclude H. pylori infection.
Objective
To investigated endoscopic mucosal lesion (Gastritis, GU, Cancer or DU) and histologic picture of gastric mucosal H. pylori infection.
Methods
This study was performed in 50 patient with gastroscopic lesion (Gastritis, GU,Cancer or DU) and CLO test positive. H. pylori was evaluated by modified Giemsa staining and gastritis by Haemotoxiline and eosin (H and E) staining in the antrum and corpus.
Result
Reported in The Third Western Pasific Helicobacter Congress, June 22‐24 Bali Indonesia.
Background
Helicobacter pylori (HP) can colonise beneath layer of mucous on the surface of gastric epithelial cell caused chronic infection more than 95% at antrum (chronic antral inflammation) besides corpus with spreading inequitably. This inflammation can cause gastric lesion as a Gastritis Gastric Ulcer (GU), Duodenal Ulcer (DU) or cancer. Absence of chronic antral inflammation (increased number of mononuclear cells in lamina propria) is a highly specific methode to exclude HP infection.
Objective
To investigated endoscopic mucosal lesion in Gastritis and histologic picture of gastric mucosal HP infection
Method
This study was performed in 50 patient with gastroscopic lesion with Gastritis and CLO test positive HP was evaluated by modified Giemsa staining and gastritis by Hematoxiline and eosin (H and E) staining in the antrum and corpus.
Result
Reported in the Third Western Pacific Helicobacter Congress, June 22‐24 Bali Indonesia.
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