Introduction: "Cerebral proliferative angiopathy" (CPA) is a distinct entity unlike "classical" brain arteriovenous malformations (AVMs). Report: We describe a patient with seizure who on DSA showed diffuse angiogenesis, absence of a well formed nidus and early venous phase suggestive of fast capillary transit favouring diagnosis of CPA alongwith filling of ophthalmic artery through middle meningeal branch of external carotid artery which made it even rarer. Treatment options are limited and vary with the patient's presentation and angiographic features. Conclusion: CPA is rare and filling of ophthalmic artery from middle meningeal artery in association with CPA is even rarer.

PIVH is rare and hypertension is important predisposing factor. Yield of cerebral angiography is high in diagnosing the etiology. AVMs and other rare etiological causes like MMD, LSA aneurysm, arterial dissection, and dAVF should be kept in mind with a high index of suspicion and warrants cerebral angiography in them, as some of the causes are potentially treatable.

Primary intraventricular haemorrhage (PIVH) is rare. Dural arteriovenous fistula causing PIVH is extremely rare. We report a case of a 17 year old boy who presented with left hemiparesis, left lower motor neuron facial palsy and ataxia. His computed tomography head revealed primary intraventricular hemorrhage. Catheter super selective angiography revealed a dural arterio venous fistula with arterial feeder arising from the middle meningeal artery as well as from the inferior marginal tentorial artery. Glue injection led to successful disappearance of the fistula and eventual clinical recovery.

A comprehensive study of favoured and unfavoured α-decay, cluster decay, weak-decay along with spontaneous fission in undetected superheavy nuclei within the range for proton number 111≤Z≤118 and neutron number 161≤N≤192 is performed. Half-lives for various mentioned decays are estimated with good accuracy on the basis of NUBASE2020 and are found in excellent match with the known half-lives. α-decay mode is found most probable in this wide range and correspondingly potential α-decay chains are reckoned. Peculiarly, the chances of cluster emission, as well as weak-decay, are also anticipated in this region of the periodic chart which open new pathways of detection of superheavy nuclei.

LGI-1 is mainly expressed in the limbic areas involving temporal cortices and hippocampus. Anti -LGI-1 encephalitis is an autoimmune encephalitis which manifests with cognitive impairment, seizures which are mostly faciobrachial dystonic type and recurrent hyponatremia. We present an 82 year old octogenarian with similar such presentation with recurrent episodes of hyponatremia and cognitive decline having an excellent response to immunotherapy. There has been no mention of a similar presentation in this age group in literature, to the best of our knowledge.