Chemotherapy-induced cardiotoxicity in adults and children is a topic with a growing interest in the cardiology literature. The ability to detect cardiac dysfunction in a timely manner is essential in order to begin adequate treatment and prevent further deterioration. This article aims to provide a review on the myocardial injury process, chemotherapeutic agents that lead to cardiotoxicity, the definition of cardiotoxicity, and the methods of timely detection and treatment.anthracyclines, cardiotoxicity, chemotherapy, congestive heart failure, pediatrics Relative to their healthy counterparts, survivors are at an eightfold higher risk of cardiovascular related deaths, including myocardial infarction with coronary artery disease, cardiomyopathy with congestive heart failure, and cerebrovascular events. 4 In childhood survivors with 30 years of follow-up, 8% had congestive heart failure. 5 These realities call for increased involvement of the pediatric cardiologist for the detection and treatment of chemotherapy-induced cardiotoxicity (CIC).The main objective of this review is to provide a background knowledge of the cause of CIC and the techniques employed to diagnose and treat CIC.
PPVI may be associated with transient VT in the acute peri-procedural period. Patients of smaller size may be more susceptible. All patients were managed conservatively and none of the patients had a recurrence, which is suggestive of a transient phenomenon.
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