Background and Objectives The study examined mirror changes in inferior wall myocardial infarction and its relationship with coronary lesions in non-infarcted areas using angiographic and electrocardiographic findings. Methods This retrospective study analyses 270 patients with inferior wall myocardial infarction, 135 of whom had mirror changes, and 135 had no. The patients' electrocardiograms, echocardiography results, and angiography results were examined. Ejection fraction, thrombolytic response, coronary artery occlusion, occlusion rate, and the number of occluded vessels were evaluated in two patients with and without mirror changes. Results We found no significant relationship between mirror changes and lateral arteries (P = 0.091). The findings indicated a connection between ST-segment depression and the existence of stenosis in the irresponsible vessel (P = 0.026). Also, in those patients exhibiting ST-segment depression, the degree of stenosis was more pronounced in the responsible vessel (P = 0.003). Additionally, no relationship was reported between multi-vessel involvement and ST-segment depression (P = 0.462). There was a noteworthy relationship between ST-segment depression and thrombolytic response (P = 0.002). Moreover, the results indicated that there was no substantial correlation between the EF level and the occurrence of ST-segment depression (P = 0.209). Conclusion It seems that mirror change in the ST segment plays a role in the severity of responsible vessel occlusion and the significance of irresponsible vessel occlusion. Mirror change did not affect EF level, though it influenced thrombolytic responses.
Background Coronary slow flow (CSF) is an angiographic entity distinguished by the delayed filling of the epicardial coronary arteries in the lack of significant obstructive artery disease. The pathological causes are still unknown. This study aimed to elucidate the relationship between clinical and laboratory-related risk factors in atherosclerosis patients diagnosed with CSF. Results The research encompassed a study group of 142 individuals, with a mean age of 52.47 ± 10.62, and a male representation of 47.7%. A thorough statistical analysis was conducted, indicating that there were no noteworthy variations in age, gender, smoking history, hematocrit, blood sugar, and HDL levels between the groups of cases and controls (P > 0.05). Subsequent analysis of the data indicated that there were significant differences in history of hypertension, LDL, and BMI measurements between the groups of subjects who were designated as cases and those who were designated as controls. Our study revealed that male gender, a history of hypertension, and BMI were identified as independent predictors of CSF (P < 0.05). Conclusions After modeling regression, we were able to conclude that male gender, BMI, and history of hypertension are reliable predictors of slow coronary flow. These findings add to our growing understanding of the complex interplay between clinical and laboratory risk factors in the development and progression of CSF.
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