Myocarditis" implies damage to heart muscle, often accompanied by impairments in heart function, as a result of inflammation caused by infection, a toxin, or immune hypersensitivity. [1][2][3] Clinical signs and symptoms (eg, fever, tachycardia, shortness of breath, myalgia, malaise), laboratory measures of damage to heart muscle (eg, creatine kinase [CK], troponin I [TROP]) and inflammation (eg, eosinophils [EOS], C-reactive protein [CRP]), and measures of heart function (eg, echocardiogram) can support the clinical diagnosis of "Presumptive Myocarditis" (PrMy) in patients. 4 Active monitoring of TROP and CRP
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