Acute post infectious cerebellitis, also known as acute cerebellar ataxia, is a rare inflammatory syndrome often characterized by cerebellar dysfunction of rapid onset. Although usually benign and self-limiting, acute cerebellitis may manifest a fulminant course. We describe a patient referred to our center with acute cerebellitis, complicated by hydrocephalus. An 32-year-old female presented with headache, nausea, vomiting, and gait and limbs ataxia 2 weeks after following a respiratory tract infection. CT showed obstructive hydrocephalus. MRI showed bilateral cerebellar swelling with hyperintense signals on T2-weighted imaging. The diagnosis was acute post infectious cerebellitis. Following treatment with corticosteroids, the clinical signs resolved. The patient made an uneventful recovery, with elimination of neurologic deficit.(JMS 2010;13(2):67-69)
BACKGROUND: Immune dysfunction is increasingly recognized to be contributing to stroke associated infections. OBJECTIVE: The present study was aimed to study the magnitude of the stroke associated infection in a mixed population of hemorrhagic and ischemic strokes and its predisposing f actors. MATERIAL &METHODS: The study population consisted of 173 consecutive stroke admissions at SKIMS Srinagar, a tertiary core hospital. The patients developing fever at admission or in hospital were included and underwent neuroimaging and workup for the source of infection. The follow up was done upto death or discharg e of the patient. A univariate and binary logistic regression analysis was done to find the patient characteristics associated with infection. RESULTS: The stroke subtypes in the study were 60.1% hemorrhagic and 39.9% ischemic strokes. The majority of hemorrhagic strokes (65A%) were located in putamina while as anterior circulation territory contributed about 80% of the ischemic strokes. Fifty eight (33.5%) patients developed stroke associated infection. Chest was the source of infection in 58.6% of the cases. The culture positivity rate was poor (18.9%) in the patient study. The presence of a comorbid illness {OR 3.4), volume of hematoma (OR 4,7). and ventricular extension of the hemorrhoge (OR 4.9) significantly increased the odds ratio of stroke associated infection. CONCLUSIONS: The frequency of post stroke infection is around 33%, chest being the commonest source. Presence of a comorbid illness, volume of hemorrhage a nd ventricular extension increase the risk of infection . JMS 2012; 15(1):22-27
Idiopathic hypertrophic pachymeningitis (IHP) is a rare, chronic nonspecific and granulomatous inflammatory disorder of the dura with unknown etiology. The diagnosis can be established by an open biopsy and exclusion of all other specific granulomatous and infectious diseases. We report a case of Idiopathic hypertrophic pachymeningitis who manifested primarily by headache and visual loss with optic atrophy. The patient was thoroughly investigated and showed no evidence of underlying cause. The diagnosis was established by an open biopsy. JMS 2011;14(1):25-27
Gingival overgrowth (GO) is a side effect, associated with some distinct classes of drugs, such as anticonvulsants, immunosuppressant, and calcium channel blockers. One of the main drugs associated with GO is the antiepileptic, diphenylhydantoin (DPH)/ phenytoin (1), which affects gingival tissues by altering extracellular matrix metabolism. Phenytoin (DPH)-induced gingival overgrowth (PIGO) due to chronic administration remains an unsolved problem especially in cases where this drug is taken without any supervision due usually to poor follow-up. Younger age groups experience more lesions than adults and in the mentally handicapped the prevalence appears to be highest. The most satisfactory treatment is the replacement of the drug by a safer antiepileptic. Patients who are to be maintained on DPH respond well to a program of meticulous oral hygiene at home along with frequent professional prophylaxes. We present a case of seizure disorder with mental retardation who developed severe gingival overgrowth wherein teeth are almost buried, caused by unsupervised intake of phenytoin. JMS 2017; 20(1):44-46
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