This paper aimed at assessing the structural performance and sustainability of cold recycled asphalt pavements. Four cold recycling technologies were investigated, including the cold centralplant recycling with emulsified and foamed asphalt binders (i.e., CCPR-E and CCPR-F), and the cold in-place recycling with emulsified and foamed asphalt binders (i.e., CIR-E and CIR-F). Firstly, the laboratory tests were conducted to comprehensively evaluate the dynamic modulus, rutting, and cracking performance of cold recycled asphalt mixtures. Subsequently, these laboratory results were used to determine the inputs of cold recycled asphalt mixtures for the Pavement ME Design program, which was employed to predict the pavement performance. Meanwhile, the National Center for Asphalt Technology also constructed four cold recycled pavement sections in the field. The monitored and predicted pavement performance showed similar trends in the first two years, but the Pavement ME Design program over predicted the rut depth of these sections. The pavement performance results confirmed that the bottom-up fatigue cracking was a negligible distress mode for cold recycled asphalt pavements. In the following, the life cycle cost analysis and life cycle assessment were conducted to evaluate the four different cold recycling projects. The life cycle cost analysis results demonstrated that all of the four cold recycling projects yielded less net present values than the HMA project. The life cycle assessment data indicated that the cold recycling technologies reduced the energy consumption by 56-64%, and decreased the greenhouse gas emissions by 39-46%. Finally, this study found that the overlay and asphalt treated base thicknesses and climatic conditions had significant impact on the performance of cold recycled asphalt pavements.
The Specific Pavement Studies Experiment 5 (SPS-5) in the Long-Term Pavement Performance program was designed to study the effects of overlay rehabilitation type on typical distress measures. The rehabilitation treatments compared overlay thickness, overlay type, and surface preparation before rehabilitation. The thicknesses used were 50- and 125-mm overlays. The overlay types were virgin asphalt mix and recycled asphalt that contained approximately 30% reclaimed asphalt pavement (RAP). Surface preparation consisted of either milling or not milling the existing pavement before rehabilitation. Eighteen states and provinces in North America built SPS-5 projects between 1989 and 1998. Seven distress parameters from these test pavements were analyzed, including international roughness index (IRI), rutting, fatigue cracking, longitudinal cracking, transverse cracking, block cracking, and raveling. Analyses were conducted to determine which factors affected overlay performance as measured with the above parameters. Further statistical testing compared the performance of the virgin mix sections directly with equivalent sections that contained 30% RAP. Overlays with mixes that contained 30% RAP were found to perform as well as overlays with virgin mixes in terms of IRI, rutting, block cracking, and raveling. Thicker overlays improved pavement performance, except for rutting. Milling before rehabilitation decreased IRI, fatigue cracking, and transverse cracking but increased rutting.
Parathyroid hormone (PTH; 10 À7 to 10 À15 M) decreased terminal chondrogenesis in the avian sterna. During the first half of an 8-day culture, 100 nM PTH (1-34) significantly increased sternal length and downregulated the deposition of type X collagen and its mRNA expression. However, it remains unclear how PTH increased cartilaginous growth. In this study, we examined growth by both cell proliferation and analysis of cyclin d1 and collagen mRNA. Types II, IX, and X collagens and cyclin d1 mRNA were quantified through real-time RT-PCR, while Ki-67 was used as an immunohistochemical proliferation marker. Extracellular matrix content was measured through mRNA quantification of types II, IX, and X collagen and observing deposition of the same collagens. PTH significantly increased the proliferation marker Ki-67 in the sternal cephalic region. There was less type II and X collagen in PTHtreated sterna with concomitant decreases in mRNA production, suggesting that proliferation was the major contributor to cartilage growth in the presence of PTH/PTH-related peptide receptor activation. In conclusion, these experiments demonstrated that PTH increased cartilage growth by upregulating cell proliferation or other extracellular matrix components. Anat Rec, 290:155-167, 2007Rec, 290:155-167, . 2007 Wiley-Liss, Inc.
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