The purpose of this review article is an overall finding from published studies on hypothermic causes of the Osborn waves induced lv
systolic dysfunction and related topics are discussed. A systematic literature search was performed in the PubMed and Embase
databases and collected 100 articles, out of which 28 were removed after peer review. We found 100 articles, out of which a total of
72 cases were considered. When analyzing only cases with more than one reported ECG, there was a strong inverse correlation
between J wave, body temperature and left ventricular dysfunction. Electrocardiographic manifestations of hypothermia may assist
in timely diagnosis and management of hypothermic patients. Even though prominent J-waves are the hallmark of hypothermic
patient’s ECG, they are not pathognomonic, as they have been associated with other inherited or acquired conditions, many of which are highly arrhythmogenic.
Hypothermia, described as a core body temperature of < 95%, is associated with ECG alteration abnormalities. Sinus bradycardia
occurs when the body temperature drops below 90°F, and is correlated with gradual prolongation of the PR interval, QRS complex,
QT interval. It can progress to ventricular and atrial fibrillation at a temperature reaching 89°F, which can lead to left ventricular
dysfunction. Hypothermia is connected to the osborn waves, which at the end of the QRS complex consist of additional deflection.
The inferior and lateral precordial leads are seen by Osborn waves, also known as J waves, Camel hump waves and hypothermic
waves. As the body temperature decreases, it becomes more pronounced and a gradual expansion of the QRS complex raises the
likelihood of ventricular fibrillation causing ventricle dysfunction.
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