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SUMMARY BackgroundUlcerative colitis (UC) and Crohn's disease (CD) can cause metabolic and inflammatory alterations.

SUMMARY BackgroundCure rates for eradication of Helicobacter pylori appear to be decreasing, thus more effective therapies must be identified.

Greater than normal production of NO in vivo is associated with lower than normal susceptibility of LDL to oxidation in vitro, suggesting that NO plays a protective role in the development of atherosclerosis.

No abstract