having a BMI #30 (p,0.017). Coronary artery calcium scores did not predict EPC growth (p=0.90). Biochemically, NADPH oxidase-2 protein expression was found to be higher in EPCs of patients with CAD compared to those without (p=0.02), but there were no significant differences in other redox proteins assessed. In EPC angiogenesis assays, male patients with CAD were found to have significantly increased migration compared to those without CAD (p=0.024). Conclusion: EPCs show differential redox signalling and migration in association with CAD, but no difference in spontaneous growth rates was detected.
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