Background and aim: Brown Sequard Syndrome (BSS) is a rare clinical presentation of incomplete spinal cord injury and even rarer caused by blunt trauma in the paediatric population. We presented a case of a paediatric patient with high cervical spinal cord injury due to blunt trauma resulting in atlantoaxial rotatory subluxation (AARS). Methods: A 10-year-old boy acquired SCI AIS D and neurological level of C4 due to AARS Fielding 1 secondary to blunt trauma. He had right extremities weakness with the majority of key muscle’s strength of MRC 2-3 and impaired sensory function under the neurological level for light touch and pinprick. The tactile impairment was more prominent on the right side with preserved proprioception, pain, and temperature sensation. Results: The patient underwent Gardner Well Tongs (GWT) traction procedure which was maintained for 2 weeks and then prescribed with Minerva Brace. He also showed signs of reflexive neurogenic bladder. The patient was hospitalised for 2 weeks in the rehabilitation ward for independence and intermittent catheterisation training. He ambulated using a wheelchair at the time of discharge but could walk independently using a quadripod cane after 1 month of follow-up, showing good motor recovery. Conclusion: Most cases of traumatic Brown Sequard usually arise from penetrating or stab wound, while blunt trauma is usually accompanied by spine fracture or non-bony elements such as ligament instability or subluxation. This patient only had AARS fielding 1, but the extensive right side motor weakness might be caused by a direct acceleration-deceleration mechanism at the cervical spinal cord followed by secondary trauma due to oedema and diminished blood perfusion. As a result, this patient did not show pure classic BSS in terms of sensory impairment but had hemiplegia. For pediatric patients, determining the prognosis and optimisation of appropriate rehabilitation programs for children is crucial for maximal outcomes and long-term quality of life.
Background and aim: Spinal tuberculosis (STB) is a hazardous type of bone tuberculosis that could affect the entire spine, with predilection towards the lower thoracic level. Upper cervical spondylitis occurred only at 0.3 – 1% of all STB cases, but this rare type leads to severe consequences, including tetraplegia, autonomic dysfunction, and respiratory impairment. This case reported long CTLSO rigid external stabilisation reasoning to boost protection whilst recovering tuberculosis despite stable internal stabilisation. Methods: A 34-year-old male came with one year of worsening low-back pain, unintentional weight loss, and gradual loss of power and sensation of both feet within a month. Physical examination showed kyphotic deformity, tenderness, key-muscle-strength 3, and sensory impairment for lower extremities, with no autonomic disturbances. Laboratory works showed chronic ongoing infection. Whole-spine MRI showed multiple spondylodiscitis with multi-focal paravertebral abscesses at C3 – C5, C7 – T3, T9 – T12, L1, severe canal stenosis and anterior segment destruction of T12. Results: The patient was diagnosed with SCI AIS D with a neurological level of L3 secondary to T12 vertebra destruction from STB. He underwent abscess evacuation, debridement, and posterior stabilisation at T10 – L3 and debridement of abscess at C3 – C5 without the need for stabilisation. After procedures, the spine was further stabilised by a CTSLO brace with flexion, extension, lateral flexion, rotation stop. Inward rehabilitation includes incremental mobilisation, upper-limb-extensor-group-strengthening-programme, balance-exercise with ambulation training. Patient discharged with status: ambulate independently with a walker. To date, the patient has returned to work with independent ambulation. Conclusion: Type-III STB was indicated for surgical procedures. Instability of upper cervical prevented patient to performed loadbearing activity thus lowering patient functional outcomes. CTLSO supplementation was given to compensate for 3 lesion sites, the upper cervical, mid-thoracic, and lower-thoracic. For the C3 – C5 lesion, there was no internal stabilisation, so a proper external mode of stabilisation should be given. However, for the thoracic level, CTLSO was given due to (1) incomplete SCI lesion in the absence of deteriorating neurological deficit,c(2) porous nature of STB, and (3) good prognostic recovery of STB with anti-TB medication leading to the reversible neurological deficit.
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