The purpose of the American College of Sports Medicine's (ACSM) exercise preparticipation health screening process is to identify individuals who may be at elevated risk for exercise-related sudden cardiac death and/or acute myocardial infarction. Recent studies have suggested that using the current ACSM exercise preparticipation health screening guidelines can result in excessive physician referrals, possibly creating a barrier to exercise participation. In addition, there is considerable evidence that exercise is safe for most people and has many associated health and fitness benefits; exercise-related cardiovascular events are often preceded by warning signs/symptoms; and the cardiovascular risks associated with exercise lessen as individuals become more physically active/fit. Consequently, a scientific roundtable was convened by the ACSM in June 2014 to evaluate the current exercise preparticipation health screening recommendations. The roundtable proposed a new evidence-informed model for exercise preparticipation health screening on the basis of three factors: 1) the individual's current level of physical activity, 2) presence of signs or symptoms and/or known cardiovascular, metabolic, or renal disease, and 3) desired exercise intensity, as these variables have been identified as risk modulators of exercise-related cardiovascular events. Identifying cardiovascular disease risk factors remains an important objective of overall disease prevention and management, but risk factor profiling is no longer included in the exercise preparticipation health screening process. The new ACSM exercise preparticipation health screening recommendations reduce possible unnecessary barriers to adopting and maintaining a regular exercise program, a lifestyle of habitual physical activity, or both, and thereby emphasize the important public health message that regular physical activity is important for all individuals.

The data demonstrate that relatively modest hypohydration ( approximately 2.7%) as a result of EID, significantly slows 5- and 10-m sprint times. Furthermore, although the glycerol hydration regimen provided a better hydration status than the placebo hydration regimen, no performance benefits were observed.

The purpose of the present investigation was to test the hypotheses that the mechanomyographic (MMG) signal would be affected by hydration status due to changes in the intra- and extracellular fluid content (which could affect the degree of fluid turbulence), changes in the filtering properties of the tissues between the MMG sensor and muscle, and changes in torque production that may accompany dehydration. Ten subjects (age 22.5 +/- 1.6 years) were tested for maximal isometric (MVC), submaximal isometric (25, 50, and 75%MVC), and maximal concentric isokinetic muscle strength of the biceps brachii in either a euhydrated or dehydrated state while the electromyographic (EMG) and MMG signals were recorded. Separate three-way and two-way ANOVAs indicated no change in torque, EMG amplitude, EMG mean power frequency (MPF), MMG amplitude, and MMG MPF with dehydration. The lack of dehydration effect suggests that MMG may be more reflective of the intrinsic contractile processes of a muscle fiber (torque production) or the motor control mechanisms (reflected by the EMG) than the tissues and fluids surrounding the muscle fiber.

In this study, we examined the relationship between serum creatine kinase activity following exercise-induced muscle damage and muscle fibre composition. Seventeen untrained males volunteered and underwent a .[Vdot]O2max test, Wingate test, and an exercise-induced muscle damage protocol. Muscle soreness and blood samples were recorded before, immediately after, and 24, 48, 72, and 96 h after exercise. Biopsy samples from the vastus lateralis were collected one week after exercise-induced muscle damage and were assessed for muscle fibre composition. There was no significant relationship (P > 0.05) between muscle fibre composition and creatine kinase activity. A significant positive correlation (P < 0.05) was observed between soreness 48 h after exercise and type II and IIb fibres, and a significant negative correlation (P < 0.05) was observed between soreness 48 h after exercise and type I muscle fibres. Significant positive correlations were observed between soreness 48 h after exercise and the fatigue index, relative average power, and relative anaerobic capacity. Our results suggest that creatine kinase activity following exercise-induced muscle damage may not be related to muscle fibre proportions, and higher post-exercise muscular pain may be related to a predominance of type II muscle fibres and higher anaerobic capabilities.

The purpose of this study was to examine changes in various aerobic and anaerobic physical performance measures in male National Collegiate Athletic Association (NCAA) Division III soccer players during the competitive soccer season. Twelve starters of the men's soccer team (mean +/- SD; age = 20.0 +/- 0.9 years, height = 175.7 +/- 8.1 cm, body mass = 73.9 +/- 11.00 kg, body mass index [BMI] 24.0 +/- 3.0 kg.m2, and percent body fat = 10.6 +/- 5.4%) were tested at the beginning (PRS) and the end (POS) of the collegiate soccer season. Each experimental trial included a maximal aerobic capacity test (VO2max); 10-, 30-, and 40-m sprints; pro-agility test; and the Wingate anaerobic power test (WAnT). From PRS to POS, VO2max significantly increased (51.05 +/- 5.97 vs. 54.64 +/- 4.90 ml.kg-1.min-1), and the 10- and 30-m sprint were significantly lower (2.03 +/- 0.15 vs. 1.96 +/- 0.11 seconds and 4.72 +/- 0.26 vs. 4.51 +/- 0.24 seconds, respectively). Anthropometric measures, 40-m sprint, pro-agility test, and WAnT were not significantly different between PRS and POS. The results of this study indicate that NCAA Division III male soccer players appear to improve aerobic and anaerobic performance measures during the competitive soccer season. It is arguable that these performance improvements may represent a poor preseason conditioning level that may result in a competitive disadvantage during the early stages of the season. An ongoing process of recruiting better-quality players that may closely follow the off-season training regimen may partially remedy this problem.