This paper describes the results of a comprehensive analysis for tsunami disaster mitigation in Padang City, Indonesia. Assessment consists of several steps, starting from the construction of tsunami hazard maps based on the most probable earthquake scenario in the future. Results are then analyzed to determine the impact on residential population along potential evacuation routes. Next, from the standpoint of hazards, we move to the analysis of human’s vulnerability during evacuation. The term “vulnerability” is associated with available evacuation time. Here, we conducted a static evacuation model using the GIS platform and a dynamic approach using multiagent paradigm. Results of evacuationmodeling suggest that some residents may not have enough time to leave the tsunami inundation area before the first wave comes. We therefore propose using relatively high buildings as vertical evacuation sites. One of potential candidates that survived from a devastated earthquake with 7.6 Mw in 2009 is selected to be further analyzed its antiseismic deficiencies based on design ground motion obtained from micro-tremor analysis and synthesized recorded wave in Padang. As a result, even though the building underwent some damage, the frame structure was able to withstand the shaking and keep the building from collapsing.
The hypersensitive response (HR) is one of the most critical defense systems in higher plants. In order to understand its molecular basis, we have screened tobacco genes that are transcriptionally activated during the early stage of the HR by the differential display method. Among six genes initially identified, one was found encoding a 57 kDa polypeptide with 497 amino acids not showing significant similarity to any reported proteins except for the AAA domain (ATPase associated with various cellular activities) spanning over 230 amino acids. The bacterially expressed protein exhibited ATP hydrolysis activity, and a green fluorescent protein-fusion protein localized in the cytoplasm of onion epidermis cells. The protein was subsequently designated as NtAAA1 (Nicotiana tabacum AAA1). NtAAA1 transcripts were induced 6 h after HR onset not only by TMV but also by incompatible Psuedomonas syringae, indicating that NtAAA1 is under the control of the N-gene with a common role in pathogen responses. Expression of NtAAA1 was induced by jasmonic acid and ethylene, but not by salicylic acid (SA). It also occurred at a high level in SA-deficient tobacco plants upon TMV infection. When NtAAA1 was silenced by the RNAi method, accumulation of transcripts for PR-1a significantly increased during the HR. Treatments with SA induced higher expression of PR-1a and acidic PR-2 in RNAi transgenic plants than in wild-type counterparts. These results suggest that NtAAA1 mitigates the SA signaling pathway, and therefore that NtAAA1 modulates the pathogen response of the host plants by adjusting the HR to an appropriate level.
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