A radiographic evaluation of the normal as well as the progressively widened tibiofibular interval in the area of the syndesmosis was done using 12 fresh cadaver lower extremities. The width of the tibiofibular "clear space" and the amount of tibiofibular overlap was determined on accurately positioned anterior-posterior and mortise radiographs. Based on a 95% confidence interval, measurements obtained for the intact specimens would support the following criteria as consistent with a normal tibiofibular relationship: (1) a tibiofibular "clear space" on the anterior-posterior and mortise views of less than approximately 6 mm; (2) tibiofibular overlap on the anterior-posterior view of greater than approximately 6 mm or 42% of fibular width; (3) tibiofibular overlap on the mortise view of greater than approximately 1 mm. The width of the tibiofibular "clear space" on both anterior-posterior and mortise views appeared to be the most reliable parameter for detecting early syndesmotic widening.
The snapping hip syndrome is a symptom complex characterized by hip pain and an audible snapping of the hip with exercise typically seen in young individuals. "External" and "internal" etiologies have been described, although the "internal" etiology is poorly understood. A clinical, radiographic, and anatomical study of eight patients with this disorder, secondary to an internal etiology, was undertaken to aid in the diagnosis and surgical treatment. Iliopsoas bursography with cineradiography revealed subluxation of the iliopsoas tendon to be an apparent cause of the snapping hip. The anatomy of the hip in relationship to the iliopsoas tendon is defined with the anterior inferior iliac spine, iliopectineal eminence, and lesser trochanter assuming a significant role in the syndrome. An operative approach involving a partial release and lengthening of the iliopsoas tendon, with minimal resection of a lesser trochanteric bony ridge, if involved, is described.
A cadaver study done to evaluate function of the deltoid ligament and its major subdivisions, the superficial and deep components, revealed that the deltoid ligament is the primary restraint against valgus tilting of the talus, with superficial and deep components being equally effective in this regard. The deep deltoid ligament appeared to be the secondary restraint against both lateral and anterior talar excursion, with the lateral malleolus and supporting ligaments being the primary restraint.
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