Introduction: Undernutrition is a major issue in the developing countries such as India. The country has the largest adolescent population in the world and one of the primary focuses of nutritional assessment among them is undernutrition. The present cross-sectional study tries to determine the prevalence of thinness among adolescent girls of Darjeeling district using thinness (BMI-for-age) and to ascertain the effects of different socio-economic and demographic variables on the same.Material and methods: The study was carried out among 387 school-going adolescent girls aged 9-14 years belonging to the Bengalee Hindu caste populations (BHCP). The prevalence of thinness was assessed using recently proposed international BMI-for-age cut-offs of Cole et al.Results: The prevalence of overall thinness was 23.77%. The distribution of mild (Grade I), moderate (Grade II), and severe (Grade III) thinness were 17.31%, 4.39% and 2.07%, respectively. The results of the binary logistic regression analysis showed that birth order, family size and father’s occupation were significantly associated with overall prevalence of thinness (p<0.05).Conclusion: The proper dissemination of awareness related to nutritional requirement, food habit, and appropriate dietary habit would be helpful to reduce the prevalence of thinness.J Nepal Paediatr Soc 2016;36(2):115-120
Aim: To assess the association of father’s education and occupation with children growth measured by height-for-age z-scores and BMI-for-age z-scores in a patriarchal culture where father’s social position is considered more important than mother’s social position. Sample and methods: The present cross-sectional study consists of 387 school-going girls aged 9-14 years residing in Matigara, Siliguri sub-division of Darjeeling district, West Bengal, India. Information on age, mother’s education, father’s education, ethnic affiliation, mother’s occupation, father’s occupation, house type, household monthly income and family size were recorded. Associations between variables were assessed using Spearman correlation, St. Nicolas house analysis (SNHA), and one-way analysis of variance (ANOVA) with box plots. Results: The hypothesis that in a patriarchy paternal socio-economic status (SES) influences children growth more than maternal SES was not supported. The observed correlation between mother education and measure of growth (BAZ and HAZ) was, 0.15 and 0.13, respectively. SNHA showed direct connection between HAZ of girls and mother education. Further, using ANOVA significant difference in the HAZ of adolescents was observed between least educated mothers and moderately educated mothers (F = 6.593; p < 0.01). No such difference between the maternal education levels was observed for BAZ. Conclusion: Maternal education is an important factor influencing children linear growth even in a patriarchy. The association was independent of nutrition. Common explanations are functional literacy, decision making, access to information and health infrastructure, and less domestic violence. Mother’s education may influence perceived future prospects of the daughters, and could be an important stimulus for growth.
Malaria is one of the major health problems in the world. It remains an important cause of very high human morbidity and mortality, especially, among children and pregnant women. It results from the infection of parasites belonging to the genus Plasmodium. Plasmodium falciparum and Plasmodium vivax are the major pathogens responsible for causing human malaria. Approximately 75% of cases are caused by P. falciparum and associated with the mortality rate of approximately 0.5 to 1.0%. Both P. falciparum and P. vivax induce anemia during their erythrocytic stages of infection. Most of the malarial infections are related to some degree of anemia, the severity of which depends upon patient-specific characteristics (e.g., age, innate and acquired resistance, comorbid features, etc.) as well as parasite-specific characteristics (e.g., species, adhesive, and drug-resistant phenotype, etc.). Malarial anemia encompasses reduced production of erythrocytes as well as increased removal of circulating erythrocytes in the bone marrow. Susceptibility to severe malarial anemia is associated with the polymorphisms of the cytokines, which are likely to function by perturbing erythropoiesis. This article reviews the epidemiology, pathophysiology, clinical features, treatment, and various complications occurring due to malarial anemia. The second part of this article also focuses on the effect of malaria during pregnancy. Some significant effects of malaria during pregnancy include spontaneous abortion, preterm delivery, low birthweight, stillbirth, congenital infection, and maternal death. How to cite this article Saxena R, Bhatia A, Midha K, Debnath M, Kaur P. Malaria: A Cause of Anemia and Its Effect on Pregnancy. World J Anemia. 2017;1(2):51-62.
The characteristics or the clinical features associated with FA have been described Table 1. Relation with Other SyndromesFanconi anemia is classified into the following group of syndromes: 8-10 • Chromosomal instability syndromes (by its genetic pathological characteristics): It includes Ataxia telangiectasia, Nijmegen breakage syndrome, Bloom syndrome, and Werner syndrome. It occurs due to the defects associated with deoxyribonucleic acid (DNA) repair mechanism, elevated cancer risk, and few other changes phenotypically. • Inherited bone marrow failure syndromes: The syndromes related to bone marrow failure are termed as the unsuccessful hematopoietic function of the bone marrow, and they include the following: Shwachman-Diamond syndrome Amegakaryocytic thrombocytopenia Pearson syndrome Dyskeratosis congenita (DKC) Diamond-Blackfan anemia Severe congenital neutropenia. Genes Involved in Fanconi AnemiaThe genome homeostasis is maintained by the FA proteins along with some other proteins. They are involved in DNA repair processes and cell division. Germ-line mutation of any one of the genes that encode FA proteins causes FA. The mutated genes found in a population with FA, are known as fine needle aspiration cytology FANC genes.
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