Topological signals defined on nodes, links and higher dimensional simplices define the dynamical state of a network or of a simplicial complex. As such, topological signals are attracting increasing attention in network theory, dynamical systems, signal processing and machine learning. Topological signals defined on the nodes are typically studied in network dynamics, while topological signals defined on links are much less explored. Here we investigate Dirac synchronization, describing locally coupled topological signals defined on the nodes and on the links of a network, and treated using the topological Dirac operator. The dynamics of signals defined on the nodes is affected by a phase lag depending on the dynamical state of nearby links and vice versa. We show that Dirac synchronization on a fully connected network is explosive with a hysteresis loop characterized by a discontinuous forward transition and a continuous backward transition. The analytical investigation of the phase diagram provides a theoretical understanding of this topological explosive synchronization. The model also displays an exotic coherent synchronized phase, also called rhythmic phase, characterized by non-stationary order parameters which can shed light on topological mechanisms for the emergence of brain rhythms.
The study of reaction-diffusion systems on networks is of paramount relevance for the understanding of nonlinear processes in systems where the topology is intrinsically discrete, such as the brain. Until now reactiondiffusion systems have been studied only when species are defined on the nodes of a network. However, in a number of real systems including, e.g., the brain and the climate, dynamical variables are not only defined on nodes but also on links, faces and higher-dimensional cells of simplicial or cell complexes, leading to topological signals. In this work we study reaction-diffusion processes of topological signals coupled through the Dirac operator. The Dirac operator allows topological signals of different dimension to interact or cross-diffuse as it projects the topological signals defined on simplices or cells of a given dimension to simplices or cells of one dimension up or one dimension down. By focusing on the framework involving nodes and links we establish the conditions for the emergence of Turing patterns and we show that the latter are never localized only on nodes or only on links of the network. Moreover when the topological signals display Turing pattern their projection does as well. We validate the theory hereby developed on a benchmark network model and on square lattices with periodic boundary conditions.
The Price model, the directed version of the Barabási–Albert model, produces a growing directed acyclic graph. We look at variants of the model in which directed edges are added to the new vertex in one of two ways: using cumulative advantage (preferential attachment) choosing vertices in proportion to their degree, or with random attachment in which vertices are chosen uniformly at random. In such networks, the longest path is well defined and in some cases is known to be a better approximation to geodesics than the shortest path. We define a reverse greedy path and show both analytically and numerically that this scales with the logarithm of the size of the network with a coefficient given by the number of edges added using random attachment. This is a lower bound on the length of the longest path to any given vertex and we show numerically that the longest path also scales with the logarithm of the size of the network but with a larger coefficient that has some weak dependence on the parameters of the model.
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