It is concluded that acute stress may produce a modest increase in alcohol consumption in healthy, nonproblem social drinkers but that this increase is not directly related to the pharmacological effects of the drug. Nonpharmacological factors may include expectancies, thirst, or nonspecific facilitation of ongoing behaviors.
These results demonstrate that acute stress can dampen subjective responses to a low dose of METH, but these effects are short-lived. It remains to be determined what mechanisms mediate this effect.
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