Peripheral nerve injuries, including plexopathy, are an important cause of disability with the neurological sequelae affecting activities of daily living and quality of life. Lumbosacral plexopathy represents a distinct entity of peripheral nerve disorders due to its relative rarity.The lumbosacral plexus represents the nerve supply to the lower back, pelvis, and legs and is derived from the ventral rami of the L1-S4 nerve roots. 1 Common etiologies for lumbosacral plexopathy including diabetes, neoplastic invasion, radiation, trauma, infection, inflammatory, infiltrative, and vascular causes like ischemia, hemorrhage, and direct compression by aortoiliac aneurysm. 1 The lumbosacral plexus has a very rich vascular supply from the five lumbar arteries that originate from the abdominal aorta, the deep circumflex iliac artery that branches from the external iliac artery, and the iliolumbar and gluteal branches of the internal iliac artery. 2 Due
An endoleak is a complication that can occur after an endovascular
aneurysm repair. We report a rare case of ischemic lumbosacral
plexopathy post embolization of type 2 endoleak, including its
presentation, neurological progress, rehabilitation strategy and
functional outcome.
Camptocormia (bent spine syndrome) is a rare complication after hemorrhagic stroke. It is a disabling, acquired postural abnormality that has a significant impact on a patient's physical function and quality of life and is more often seen in patients with Parkinson's disease. Treatment strategies pertaining to this condition can be broadly divided into invasive which involved deep brain stimulation and spinal fixation operation versus non-invasive approaches like physiotherapy, orthosis and drugs. However, most of the treatments described in the past are mainly for Parkinson's patients with camptocormia, and none are for camptocormia from hemorrhagic stroke. We report a rare case of camptocormia in a posthemorrhagic stroke patient whose rehabilitation progress was greatly impeded by this axial postural deformity and who responded well to Madopar treatment with an improvement in total camptocormia angle from 90 degrees to about 30 degrees, which translated to an improvement in physical function and reduction in care burden.
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